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Alberto Rábano
Researcher at Carlos III Health Institute
Publications - 107
Citations - 4928
Alberto Rábano is an academic researcher from Carlos III Health Institute. The author has contributed to research in topics: Medicine & Dementia. The author has an hindex of 31, co-authored 86 publications receiving 3366 citations. Previous affiliations of Alberto Rábano include Spanish National Research Council.
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Journal ArticleDOI
Evidence for fungal infection in cerebrospinal fluid and brain tissue from patients with amyotrophic lateral sclerosis.
Ruth Alonso,Diana Pisa,Ana Isabel Marina,Esperanza Morato,Alberto Rábano,Izaskun Rodal,Luis E. Carrasco +6 more
TL;DR: Observations provide compelling evidence of fungal infection in the ALS patients analyzed, suggesting that this infection may play a part in the etiology of the disease or may constitute a risk factor for these patients.
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Polymicrobial Infections In Brain Tissue From Alzheimer’s Disease Patients
TL;DR: The results show that polymicrobial infections consisting of fungi and bacteria can be revealed in brain tissue from AD patients.
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Direct visualization of fungal infection in brains from patients with Alzheimer's disease.
TL;DR: Fungal infection can only be observed using specific anti-fungal antibodies and only a small percentage of cells contain fungi, consistent with the idea that fungal cells are internalized inside neurons in AD patients.
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ApoE gene and exceptional longevity: Insights from three independent cohorts
Nuria Garatachea,Nuria Garatachea,Enzo Emanuele,Miguel Calero,Noriyuki Fuku,Yasumichi Arai,Yukiko Abe,Haruka Murakami,Motohiko Miyachi,Thomas Yvert,Zoraida Verde,Ma Ascensión Zea,Letizia Venturini,Catalina Santiago,Alejandro Santos-Lozano,Gabriel Rodríguez-Romo,Giovanni Ricevuti,Nobuyoshi Hirose,Alberto Rábano,Alejandro Lucia +19 more
TL;DR: The data suggest that the ApoE might be a candidate to influence EL, and the ε4-allele appears to decrease the likelihood of reaching EL among individuals of different ethnic/geographic origins.
Journal ArticleDOI
Tau hyperphosphorylation induces oligomeric insulin accumulation and insulin resistance in neurons
Patricia Rodriguez-Rodriguez,Anna Sandebring-Matton,Paula Merino-Serrais,Cristina Parrado-Fernandez,Alberto Rábano,Bengt Winblad,Jesús Avila,Jesús Avila,Isidre Ferrer,Isidre Ferrer,Angel Cedazo-Minguez +10 more
TL;DR: It is reported that insulin is accumulated and retained as oligomers in hyperphosphorylated tau-bearing neurons in Alzheimer's disease and in several of the most prevalent human tauopathies, and a novel neuropathological concept with important therapeutic implications is described.