scispace - formally typeset
A

Alexandra Giatromanolaki

Researcher at Democritus University of Thrace

Publications -  360
Citations -  26647

Alexandra Giatromanolaki is an academic researcher from Democritus University of Thrace. The author has contributed to research in topics: Angiogenesis & Cancer. The author has an hindex of 66, co-authored 340 publications receiving 22956 citations. Previous affiliations of Alexandra Giatromanolaki include John Radcliffe Hospital.

Papers
More filters
Journal Article

Endometrial carcinoma and diabetes revisited.

TL;DR: Results indicate that endometrial adenocarcinoma is not intrinsically different in diabetic patients, but long-term survival and other clinical and histological features were the same in the two groups.
Journal ArticleDOI

Differential assessment of angiogenic activity and of vascular survival ability (VSA) in breast cancer.

TL;DR: Recognition and quantification of the vascular survival ability in human tumors may significantly improve the prognostic value of the assessment of tumor vasculature, and may help to stratify patients for clinical trials with novel anti-angiogenic or angiotoxic drugs.
Journal ArticleDOI

Inferior nasal turbinate wound healing after submucosal radiofrequency tissue ablation and monopolar electrocautery: histologic study in a sheep model.

TL;DR: To study differences in inferior nasal turbinate mucosal and submucosal wound‐healing histology after radiofrequency tissue ablation (RTA) and monopolar electrocautery (MEC), data are collected on mice treated with RTA and MEC.
Journal ArticleDOI

Interferon regulatory factor-1 (IRF-1) suppression and derepression during endometrial tumorigenesis and cancer progression.

TL;DR: It is concluded that down-regulation of IRF-1 is a constant finding in endometrial tumorigenesis, however, derepression of IRf-1 may occur in a subset of tumors, and this event is associated with TP up-regulation and aggressive tumor behavior.
Journal ArticleDOI

Inhibition of IKK-NFκB pathway sensitizes lung cancer cell lines to radiation.

TL;DR: The genetic or pharmacological blockage of the RAF1-ERK-IKK-NFκB pathway sensitizes cells to therapeutic doses of radiation, and the IKK pathway is a promising target for therapeutic intervention in combination with radiotherapy.