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Ambereen Ali

Researcher at Duke University

Publications -  5
Citations -  760

Ambereen Ali is an academic researcher from Duke University. The author has contributed to research in topics: DNA damage & Cell cycle. The author has an hindex of 5, co-authored 5 publications receiving 685 citations. Previous affiliations of Ambereen Ali include University of Malaya.

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Journal ArticleDOI

ATR/ATM-mediated phosphorylation of human Rad17 is required for genotoxic stress responses.

TL;DR: It is demonstrated that ATR/ATM-dependent phosphorylation of hRad17 is a critical early event during checkpoint signalling in DNA-damaged cells.
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Requirement of protein phosphatase 5 in DNA-damage-induced ATM activation

TL;DR: PP5 plays an essential role in the activation and checkpoint signaling functions of ATM in cells that have suffered DNA double-strand breaks, and a direct regulatory linkage between PP5 and ATM is reported.
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Prehospital transdermal glyceryl trinitrate in patients with ultra-acute presumed stroke (RIGHT-2): an ambulance-based, randomised, sham-controlled, blinded, phase 3 trial

Philip M.W. Bath, +417 more
- 09 Mar 2019 - 
TL;DR: Prehospital treatment with transdermal GTN does not seem to improve functional outcome in patients with presumed stroke, and it is feasible for UK paramedics to obtain consent and treat patients with stroke in the ultra-acute prehospital setting.
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Effect of a palm-oil-vitamin E concentrate on the serum and lipoprotein lipids in humans.

TL;DR: The results show that the palmvitee has a hypocholesterolemic effect and lowered both serum total cholesterol (TC) and low-density-lipoprotein cholesterol (LDL-C) concentrations in all the volunteers.
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Protein Phosphatase 5 Is Required for ATR-Mediated Checkpoint Activation

TL;DR: It is demonstrated that protein phosphatase 5 (PP5) is required for the ATR-mediated checkpoint activation and reduced PP5 activity exerts differential effects on the formation of intranuclear foci by ATR and replication protein A, implicating a functional role for PP5 in a specific stage of the checkpoint signaling pathway.