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Anil Chandraker

Researcher at Brigham and Women's Hospital

Publications -  225
Citations -  11075

Anil Chandraker is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Transplantation & Kidney transplantation. The author has an hindex of 51, co-authored 215 publications receiving 9892 citations. Previous affiliations of Anil Chandraker include Lahey Hospital & Medical Center & National Institutes of Health.

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Journal Article

Inhibition of Simian virus 40 large T antigen helicase activity by fluoroquinolones.

TL;DR: Flinoroquinolones and their derivates may be useful in the treatment and/or prevention of infection by SV40-homologous human DNA viruses that encode helicase activity for their survival.
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Fasting Plasma Total Homocysteine Levels and Mortality and Allograft Loss in Kidney Transplant Recipients: A Prospective Study

TL;DR: In this single-center sample, baseline fasting plasma tHcy levels were independently associated with the risk of death and kidney allograft loss and the clinical utility of homocysteine-lowering therapy, such as multivitamin therapy, to reduce the rates of these end points needs to be studied.
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Impact of Pretransplant Anti-HLA Antibodies on Outcomes in Lung Transplant Candidates

TL;DR: The presence of anti-HLA antibodies at the high MFI threshold (>3,000) was associated with lower transplant rate and higher rates of AMR, and screening for anti- HLA antibodies using the 3,000 M FI threshold may be important in managing transplant candidates and recipients.
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Invasive fungal infections and antifungal therapies in solid organ transplant recipients

TL;DR: Determining the best course of therapy is difficult due to the limited availability of data in SOT recipients and much of the available information is often based on case‐reports or retrospective analyses.
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Essential Role of PDL1 Expression on Nonhematopoietic Donor Cells in Acquired Tolerance to Vascularized Cardiac Allografts

TL;DR: It is demonstrated that PDL1 expression mainly on donor endothelium is functionally important in a fully allogeneic mismatched model for the induction of cardiac allograft tolerance.