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James A. DeCaprio

Researcher at Harvard University

Publications -  236
Citations -  25368

James A. DeCaprio is an academic researcher from Harvard University. The author has contributed to research in topics: Merkel cell carcinoma & Retinoblastoma protein. The author has an hindex of 75, co-authored 217 publications receiving 23432 citations. Previous affiliations of James A. DeCaprio include Institute of Cancer Research & Dana Corporation.

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SV40 large tumor antigen forms a specific complex with the product of the retinoblastoma susceptibility gene

TL;DR: Results are consistent with a model for transformation by SV40 which, at least in part, involves T/p110-114 complex formation and the perturbation of Rb protein and/or T function.
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Molecular cloning and functional analysis of the adenovirus E1A-associated 300-kD protein (p300) reveals a protein with properties of a transcriptional adaptor.

TL;DR: It is shown that p300 molecules lacking an intact E1A-binding site can bypass E 1A repression and restore to a significant extent the activity of the SV40 enhancer, even in the presence of high levels of E1a protein.
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The product of the retinoblastoma susceptibility gene has properties of a cell cycle regulatory element.

TL;DR: The cell cycle-dependent phosphorylation of Rb is demonstrated and a model to explain how Rb may suppress cell growth by acting as a cell cycle regulatory element is proposed.
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Expression cloning of a cDNA encoding a retinoblastoma-binding protein with E2F-like properties

TL;DR: It is found that RBAP-1 copurifies with E2F, interacts specifically with the adenovirus E4 ORF 6/7 protein, binds specifically and directly to a known E2f DNA recognition sequence, and contains a functional tranasactivation domain.
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Growth inhibition by TGF-β linked to suppression of retinoblastoma protein phosphorylation

TL;DR: TGF- beta 1 and RB appear to function in a common growth-inhibitory pathway in which TGF-beta 1 acts to retain RB in the underphosphorylated, growth-suppressive state.