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Anil Chandraker
Researcher at Brigham and Women's Hospital
Publications - 225
Citations - 11075
Anil Chandraker is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Transplantation & Kidney transplantation. The author has an hindex of 51, co-authored 215 publications receiving 9892 citations. Previous affiliations of Anil Chandraker include Lahey Hospital & Medical Center & National Institutes of Health.
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Journal ArticleDOI
New England BK consortium: Regional survey of BK screening and management protocols in comparison to published consensus guidelines.
Steven Gabardi,Steven Gabardi,Martha Pavlakis,Chen Tan,Chen Tan,Jean M. Francis,Jean M. Francis,Francesca Cardarelli,William S. Asch,Kenneth A. Bodziak,Kenneth A. Bodziak,Michael Chobanian,Michael Chobanian,Hannah Gilligan,Reginald Y. Gohh,Reginald Y. Gohh,Shiang-Cheng Kung,Shiang-Cheng Kung,Lesley A. Inker,Lesley A. Inker,Spencer T. Martin,Nancy Rodig,Ana P. Rossi,Anil Chandraker,Anil Chandraker +24 more
TL;DR: The New England BK Consortium aims to jointly optimize screening and management of BKPyV, which continues to impact renal transplant recipients (RTR).
Journal ArticleDOI
Role of Regulatory T Cells in the “Infectious” Tolerance Pathway in Transplant Recipients
K. Onodera,K. Onodera,Anil Chandraker,Hans-Dieter Volk,Manfred Lehmann,S. Korom,T.H.W Stadlbauer,Kazuya Kato,S Kasai,Mohamed H. Sayegh,Jerzy W. Kupiec-Weglinski +10 more
Journal ArticleDOI
Regulatory t cells maintain peripheral tolerance to islet allografts induced by intrathymic injection of mhc class i allopeptides
David V. Saborio,Nepal C. Chowdhury,Ming-Xing Jin,Mohamed H. Sayegh,Anil Chandraker,M. A. Hardy,S.F Oluwole +6 more
TL;DR: The finding that IT injection of a short segment of a synthetic immunodominant MHC class I peptide derived from WAG that shares the RT1.A(U) domain with the graft donor is capable of inducing acquired systemic tolerance to WF islets suggests that linked recognition or epitope suppression may be involved in the induction of unresponsiveness.
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Donor myeloid derived suppressor cells (MDSCs) prolong allogeneic cardiac graft survival through programming of recipient myeloid cells in vivo
Songjie Cai,Songjie Cai,John Choi,Thiago J. Borges,Hengcheng Zhang,Ji Miao,Takaharu Ichimura,Xiaofei Li,Simiao Xu,Philip Chu,Siawosh K. Eskandari,Hazim Allos,Juliano B. Alhaddad,Saif A. Muhsin,Karim M. Yatim,Leonardo V. Riella,Peter T. Sage,Anil Chandraker,Jamil Azzi +18 more
TL;DR: Donor-derived M DSCs prolong cardiac allograft survival in a donor-specific manner via induction of recipient’s endogenous MDSCs and observed that the allogeneic mixed lymphocytes reaction was suppressed in the presence of CD11b+Gr1+ MDSC in a donors’specific manner.
Journal ArticleDOI
The Limits of Linked Suppression for Regulatory T Cells
Toshiro Ito,Akira Yamada,Ibrahim Batal,Melissa Y. Yeung,Martina M. McGrath,Mohamed H. Sayegh,Anil Chandraker,Takuya Ueno +7 more
TL;DR: The limits of linkage appear to be quantitative and not universally determined by either the indirect pathway or by peptides of donor MHC Ags.