Anilton Cesar Vasconcelos

TL;DR: These findings provide direct evidence that decreased cholinergic neurotransmission and underlying autonomic imbalance cause plastic alterations that contribute to heart dysfunction.

TL;DR: This is the first in vivo-administered toxicant to be described that targets myoid cells, resulting in abnormal spermatogenesis.

TL;DR: The role of type I IFN signaling during Brucella abortus infection, a facultative intracellular bacterial pathogen that causes abortion in domestic animals and undulant fever in humans, is addressed and it is demonstrated that IFN-αβR KO mice are more resistant to infection suggesting that type IIFN signaling is detrimental to host control of Bru cell control.

TL;DR: The results indicate that during TD, reactive oxygen species (ROS) production may be enhanced as a consequence of the installed acidosis and the perturbation in the cardiac myocytes redox balance was responsible for the increase in apoptosis.

TL;DR: Retropharingeal lymph nodes of naturally and experimentally infected dogs had more apoptotic cells per field than controls and DNA from thymus of infected dogs were more fragmented than controls, suggesting apoptosis is increased in lymphoid depletion induced by canine distemper virus and consequently play a role in the immunosuppression seen in this disease.