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Antonino Nicoletti
Researcher at University of Paris
Publications - 162
Citations - 10149
Antonino Nicoletti is an academic researcher from University of Paris. The author has contributed to research in topics: Immune system & Antigen. The author has an hindex of 47, co-authored 151 publications receiving 9157 citations. Previous affiliations of Antonino Nicoletti include Pierre-and-Marie-Curie University & Paris Descartes University.
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The vascular smooth muscle cell in arterial pathology: a cell that can take on multiple roles.
TL;DR: The medial VSMC is the organizer of the inwardly directed angiogenic response arising from the adventitia by overexpressing vascular endothelial growth factor in response to lipid-stimulated peroxisome proliferator-activated receptor-γ, and probably also the organizers of the adventitial immune response by secreting chemokines.
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Transfer of CD4+ T Cells Aggravates Atherosclerosis in Immunodeficient Apolipoprotein E Knockout Mice
TL;DR: CD4+ T cells carry disease-promoting immunity in atherosclerosis and are associated with the infiltration of transferred T cells into lesions, increased circulating interferon-&ggr; levels, and increased I-A expression in lesions.
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Protective immunity against atherosclerosis carried by B cells of hypercholesterolemic mice
TL;DR: It is shown that spleen-associated immune activity protects against atherosclerosis and reduces disease progression, and a role for T-B cell cooperation is suggested.
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Angiotensin II stimulates endothelial vascular cell adhesion molecule-1 via nuclear factor-kappaB activation induced by intracellular oxidative stress.
Maria E. Pueyo,Walter Gonzalez,Antonino Nicoletti,Françoise Savoie,Jean-François Arnal,Jean-Baptiste Michel +5 more
TL;DR: It is demonstrated that Ang II induced an intracellular oxidative stress in endothelial cells, which stimulates IkappaB degradation and nuclear factor-kappaB activation and enhances the expression of VCAM-1 and probably other genes involved in the early stages of atherosclerosis.
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Macrophage Plasticity in Experimental Atherosclerosis
Jamila Khallou-Laschet,Aditi Varthaman,Giulia Fornasa,Caroline Compain,Anh Thu Gaston,Marc Clement,Michael Dussiot,Olivier Levillain,Stéphanie Graff-Dubois,Stéphanie Graff-Dubois,Antonino Nicoletti,Giuseppina Caligiuri +11 more
TL;DR: The study suggests that interventional tools able to revert the MØ infiltrate towards the M2 phenotype may exert an atheroprotective action.