B
B.J. Kullberg
Researcher at Radboud University Nijmegen
Publications - 97
Citations - 3791
B.J. Kullberg is an academic researcher from Radboud University Nijmegen. The author has contributed to research in topics: Cytokine & Proinflammatory cytokine. The author has an hindex of 34, co-authored 97 publications receiving 3587 citations.
Papers
More filters
Journal ArticleDOI
ESCMID guideline for the diagnosis and management of Candida diseases 2012:diagnostic procedures
Manuel Cuenca-Estrella,Paul E. Verweij,Maiken Cavling Arendrup,S. Arikan-Akdagli,Jacques Bille,J.P. Donnelly,Henrik Jeldtoft Jensen,Cornelia Lass-Flörl,Malcolm Richardson,Murat Akova,Matteo Bassetti,Thierry Calandra,Elio Castagnola,Oliver A. Cornely,Jorge Garbino,Andreas H. Groll,Raoul Herbrecht,William W. Hope,B.J. Kullberg,O. Lortholary,O. Lortholary,Wouter Meersseman,George Petrikkos,Emmanuel Roilides,Claudio Viscoli,Andrew J. Ullmann +25 more
TL;DR: In this paper, a panel of experts of the European Fungal Infection Study Group (EFISG) undertook a data review and compiled guidelines for the clinical utility and accuracy of different diagnostic tests and procedures for detection of Candida infections.
Journal ArticleDOI
Increased Production of Interleukin 4 by CD4+ and CD8+ T Cells from Patients with Tuberculosis Is Related to the Presence of Pulmonary Cavities
R. van Crevel,Elvina Karyadi,F. Preyers,M. Leenders,B.J. Kullberg,R.H.H. Nelwan,J.W.M. van der Meer +6 more
TL;DR: The Th2-type response with increased production of IL-4 in patients with tuberculosis may antagonize host defense and lead to tissue necrosis, and little is known about their role in tissue damage.
Journal ArticleDOI
Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections.
Mihai G. Netea,P.N.M. Demacker,B.J. Kullberg,O.C. Boerman,Ineke Verschueren,A.F.H. Stalenhoef,J.W.M. van der Meer +6 more
TL;DR: Exogenous LDL can protect against the lethal effects of endotoxin and Gram-negative infection, at least part of this protection is achieved through decreased in vivo production of pro-inflammatory cytokines, in spite of increased cytokine production capacity.
Journal ArticleDOI
Recognition of fungal pathogens by Toll-like receptors
TL;DR: New data suggest that TLRs offer escape mechanisms to certain pathogenic microorganisms, especially through TLR2-driven induction of anti-inflammatory cytokines, and the study of TLRs remains one of the most active areas of research in the field of fungal infections.
Journal Article
Increased susceptibility of TNF-alpha lymphotoxin-alpha double knockout mice to systemic candidiasis through impaired recruitment of neutrophils and phagocytosis of Candida albicans.
M.G. Netea,L. J. H. Van Tits,J.H.A.J. Curfs,Franck Amiot,Jacques F. Meis,J.W.M. van der Meer,B.J. Kullberg +6 more
TL;DR: In conclusion, endogenous TNF and/or LT contribute to host resistance to disseminated candidiasis, and their absence in TNF-/-LT-/- mice renders the animals susceptible through impaired recruitment of neutrophils and impaired phagocytosis of C. albicans.