B
Badri N. Pandey
Researcher at Homi Bhabha National Institute
Publications - 110
Citations - 3009
Badri N. Pandey is an academic researcher from Homi Bhabha National Institute. The author has contributed to research in topics: Apoptosis & Cancer. The author has an hindex of 28, co-authored 106 publications receiving 2511 citations. Previous affiliations of Badri N. Pandey include Bhabha Atomic Research Centre & Rutgers University.
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Relevance of radiobiological concepts in radionuclide therapy of cancer.
TL;DR: A few studies performed using in vitro and in vivo models envisage the possibilities of exploiting the radiobiological knowledge for improved therapeutic outcome of RNT.
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Magnetic nanoparticle-mediated hyperthermia therapy induces tumour growth inhibition by apoptosis and Hsp90/AKT modulation
Neena G. Shetake,Amit Kumar,Snehal M. Gaikwad,Pritha Ray,Sejal Desai,Raghumani Singh Ningthoujam,Rajesh K. Vatsa,Badri N. Pandey +7 more
TL;DR: These results suggest the involvement of apoptosis and Hsp90/AKT modulation in MN-OA-mediated MHT-induced tumour growth inhibition.
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Glutamic acid-coated Fe3O4 nanoparticles for tumor-targeted imaging and therapeutics.
Bijaideep Dutta,Anshika Nema,Neena G. Shetake,Jagriti Gupta,K.C. Barick,K.C. Barick,Malini A. Lawande,Badri N. Pandey,Indira K. Priyadarsini,Puthusserickal A. Hassan,Puthusserickal A. Hassan +10 more
TL;DR: The dual drugs loaded system (DOX-MTX-GAMNPs) shows pH dependent release behaviour of both the drugs and enhanced toxicity towards breast cancer cell line (MCF-7) as compared to their individual treatment.
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Role of membrane sialic acid and glycophorin protein in thorium induced aggregation and hemolysis of human erythrocytes.
TL;DR: Human erythrocytes, as a classical cellular membrane model, were coincubated withThorium-232 ((232)Th) in order to elucidate whether this naturally occurring important radionuclide produced perturbations to cell membrane, and revealed that ery throatcytes underwent aggregation or lysis depending on the ratio of (232) Th to cell.
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In vitro studies on radiation induced membrane oxidative damage in apoptotic death of mouse thymocytes
TL;DR: Results indicate that the enhanced amount of intracellular reactive oxygen species generated in response to radiooxidative stress were linked with the induction of γ irradiation of apoptotic death in thymocytes.