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Badri N. Pandey

Researcher at Homi Bhabha National Institute

Publications -  110
Citations -  3009

Badri N. Pandey is an academic researcher from Homi Bhabha National Institute. The author has contributed to research in topics: Apoptosis & Cancer. The author has an hindex of 28, co-authored 106 publications receiving 2511 citations. Previous affiliations of Badri N. Pandey include Bhabha Atomic Research Centre & Rutgers University.

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In vivo space radiation-induced non-targeted responses: late effects on molecular signaling in mitochondria.

TL;DR: Novel data is shown on the induction of in vivo non-targeted effects following exposure to 1100 MeV/nucleon titanium ions and network analyses demonstrate that 'network analyses' is an effective tool towards characterizing the signaling pathways that mediate the long-term biological effects of space radiation.
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Radiobiological basis in management of accidental radiation exposure

TL;DR: The present review discusses the knowledge gained in radiation biology that can be exploited for better treatment and management of radiation accident victims and deeper insight into the mechanisms of radiation injury under accidental radiation conditions would be helpful in achieving better biodosimetry, decorporation strategies and improvement in prevention/post-irradiation management of Radiation accident patients.
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The interaction of actinide and lanthanide ions with hemoglobin and its relevance to human and environmental toxicology

TL;DR: A biochemical basis is provided to understand the actinide/lanthanide-induced interference in heme, which may have significant implications for the medical and environmental management of lanthanides/actinides toxicity.
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Synthesis and characterization of monodispersed water dispersible Fe 3 O 4 nanoparticles and in vitro studies on human breast carcinoma cell line under hyperthermia condition

TL;DR: Heat generation from these MNPs for hyperthermia application under alternating current (AC) magnetic field is demonstrated and optimized heating efficiency by controlling morphology of particles is optimized.
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Damaging and protective bystander cross-talk between human lung cancer and normal cells after proton microbeam irradiation.

TL;DR: Investigating the bystander cross-talk between lung cancer (A549) and normal (WI38) cells after proton-microbeam irradiation using γ-H2AX foci fluorescence as a measure of DNA double-strand breaks (DSBs) observed bystander signalling between tumour and normal cells may have potential implications in therapeutic outcome of cancer radiotherapy.