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Bernd W. Scheithauer

Researcher at Mayo Clinic

Publications -  729
Citations -  58507

Bernd W. Scheithauer is an academic researcher from Mayo Clinic. The author has contributed to research in topics: Adenoma & Pituitary adenoma. The author has an hindex of 119, co-authored 729 publications receiving 55985 citations. Previous affiliations of Bernd W. Scheithauer include University of North Dakota & University of Michigan.

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Correlation of cytogenetic analysis and loss of heterozygosity studies in human diffuse astrocytomas and mixed oligo-astrocytomas.

TL;DR: The genetic analyses of these tumors suggest that there are 2 regions on chromosome 10 that may contain potential tumor suppressor genes and the results suggest that cytogenetic analysis was more sensitive in detecting an abnormality than molecular genetic studies.
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Results of surgical treatment for growth hormone-secreting pituitary adenomas

TL;DR: Surgical excision of a pituitary adenoma is the most effective therapy currently available for acromegaly and 90 of 174 patients treated between 1972 and 1983 were in remission.
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Symptomatic glial cysts of the pineal gland

TL;DR: The authors stresses the importance of recognizing the histopathological spectrum of pineal cysts, as well as correlation with radiographic findings, if a correct diagnosis is to be attained.
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PI3K/AKT pathway alterations are associated with clinically aggressive and histologically anaplastic subsets of pilocytic astrocytoma

TL;DR: In this paper, the activation of PI3K/AKT in addition to MAPK/ERK signaling pathways may underlie biological aggressiveness in PA, which may mediate the increased proliferative activity observed in histologically anaplastic PA.
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Oligodendrocyte Injury Is an Early Event in Lesions of Multiple Sclerosis

TL;DR: The ultrastructural features of 11 stereotaxic brain biopsy specimens that demonstrated inflammatory primary demyelination consistent with acute multiple sclerosis were examined and disturbances of the myelinating function of oligodendrocytes may be among the earliest pathologic features in multiple sclerosis.