B
Bogdan Panaitescu
Researcher at National Institutes of Health
Publications - 67
Citations - 2679
Bogdan Panaitescu is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Amniotic fluid & Chorioamnionitis. The author has an hindex of 28, co-authored 64 publications receiving 1901 citations. Previous affiliations of Bogdan Panaitescu include University of Alberta & Boston Children's Hospital.
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Journal ArticleDOI
Metformin, the aspirin of the 21st century: its role in gestational diabetes mellitus, prevention of preeclampsia and cancer, and the promotion of longevity.
Roberto Romero,Offer Erez,Offer Erez,Offer Erez,Maik Hüttemann,Eli Maymon,Eli Maymon,Eli Maymon,Bogdan Panaitescu,Agustin Conde-Agudelo,Percy Pacora,Percy Pacora,Bo Hyun Yoon,Lawrence I. Grossman +13 more
TL;DR: The mechanisms by which metformin may prevent preeclampsia include a reduction in the production of antiangiogenic factors and the improvement of endothelial dysfunction, probably through an effect on the mitochondria.
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The frequency and type of placental histologic lesions in term pregnancies with normal outcome.
Roberto Romero,Yeon Mee Kim,Yeon Mee Kim,Percy Pacora,Percy Pacora,Chong Jai Kim,Chong Jai Kim,Neta Benshalom-Tirosh,Neta Benshalom-Tirosh,Sunil Jaiman,Sunil Jaiman,Gaurav Bhatti,Jung Sun Kim,Jung Sun Kim,Faisal Qureshi,Faisal Qureshi,Suzanne M. Jacques,Suzanne M. Jacques,Eun Jung Jung,Eun Jung Jung,Lami Yeo,Lami Yeo,Bogdan Panaitescu,Bogdan Panaitescu,Eli Maymon,Eli Maymon,Eli Maymon,Sonia S. Hassan,Sonia S. Hassan,Chaur-Dong Hsu,Offer Erez,Offer Erez,Offer Erez +32 more
TL;DR: Most placentas had lesions consistent with inflammatory or vascular lesions, but severe and/or high-burden lesions were infrequent, and Mild placental lesions may be interpreted as acute changes associated with parturition or as representative of a subclinical pathological process that did not affect the clinical course of pregnancy.
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Evidence that intra-amniotic infections are often the result of an ascending invasion - a molecular microbiological study.
Roberto Romero,Nardhy Gomez-Lopez,Andrew D. Winters,Andrew D. Winters,Eun Jung Jung,Eun Jung Jung,Majid Shaman,Majid Shaman,Janine Bieda,Janine Bieda,Bogdan Panaitescu,Bogdan Panaitescu,Percy Pacora,Percy Pacora,Offer Erez,Offer Erez,Offer Erez,Jonathan M. Greenberg,Madison M. Ahmad,Chaur-Dong Hsu,Kevin R. Theis,Kevin R. Theis +21 more
TL;DR: Findings indicate that ascension from the lower genital tract is the primary pathway for intra-amniotic infection.
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Intra-Amniotic Administration of HMGB1 Induces Spontaneous Preterm Labor and Birth.
Nardhy Gomez-Lopez,Nardhy Gomez-Lopez,Roberto Romero,Olesya Plazyo,Olesya Plazyo,Bogdan Panaitescu,Amy Eunice Furcron,Amy Eunice Furcron,Derek Miller,Derek Miller,Tamara Roumayah,Emily Flom,Sonia S. Hassan,Sonia S. Hassan +13 more
TL;DR: The aim of this study was to determine whether intra‐amniotic administration of an alarmin, HMGB1, could induce preterm labor/birth.
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Are amniotic fluid neutrophils in women with intraamniotic infection and/or inflammation of fetal or maternal origin?
Nardhy Gomez-Lopez,Nardhy Gomez-Lopez,Roberto Romero,Yi Xu,Yi Xu,Yaozhu Leng,Yaozhu Leng,Valeria Garcia-Flores,Derek Miller,Derek Miller,Suzanne M. Jacques,Suzanne M. Jacques,Sonia S. Hassan,Sonia S. Hassan,Jonathan Faro,Adham Alsamsam,Ali Alhousseini,Hunter Gomez-Roberts,Bogdan Panaitescu,Bogdan Panaitescu,Lami Yeo,Lami Yeo,Eli Maymon,Eli Maymon +23 more
TL;DR: Evidence is provided that both fetal and maternal neutrophils can invade the amniotic cavity, suggesting that both the fetus and the mother participate in the host defense mechanisms against intraamniotic infection.