B
Brian Boag
Researcher at James Hutton Institute
Publications - 31
Citations - 1467
Brian Boag is an academic researcher from James Hutton Institute. The author has contributed to research in topics: Population & Rabbit haemorrhagic disease. The author has an hindex of 18, co-authored 31 publications receiving 1290 citations. Previous affiliations of Brian Boag include University of Glasgow.
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Competition and mutualism among the gut helminths of a mammalian host
TL;DR: Evidence of consistent interspecific interactions in a natural mammalian system is presented through the analysis of parasite intensity data collected from a free-ranging rabbit population, sampled monthly for a period of 23 yr, suggesting that parasite interactions could have profound implications for the dynamics of parasite communities.
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Global change, parasite transmission and disease control: lessons from ecology
Joanne Cable,Iain Barber,Brian Boag,Amy Ellison,Eric R. Morgan,Kris A. Murray,Emily L. Pascoe,Steven M. Sait,Anthony J. Wilson,Mark Booth +9 more
TL;DR: Evidence regarding potential effects of ‘system changes’ on parasite transmission from wild host–parasite systems is highlighted and synthesized to support robust predictions of altered parasite dynamics in a rapidly changing world.
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Peak shift and epidemiology in a seasonal host–nematode system
TL;DR: The dynamics of infection of the nematode Trichostrongylus retortaeformis within a natural population of rabbits sampled monthly for 26 years are examined, supporting the principle that acquired immunity can be modelled using the cumulative exposure to infection.
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Parasite co-infection and interaction as drivers of host heterogeneity.
TL;DR: It is concluded that concomitant infections mediated by host immunity are important in modifying host susceptibility and influencing heterogeneity amongst individual hosts.
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Climate disruption and parasite-host dynamics: patterns and processes associated with warming and the frequency of extreme climatic events.
TL;DR: It is speculated that climate disruption will lead to increased frequency and intensity of disease outbreaks in parasite populations not regulated by acquired immunity.