B
Brian W. Booth
Researcher at Clemson University
Publications - 46
Citations - 1470
Brian W. Booth is an academic researcher from Clemson University. The author has contributed to research in topics: Stem cell & Cancer. The author has an hindex of 17, co-authored 42 publications receiving 1303 citations. Previous affiliations of Brian W. Booth include National Institutes of Health & North Carolina State University.
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Interaction with the mammary microenvironment redirects spermatogenic cell fate in vivo
TL;DR: It is shown that cells from adult seminiferous tubules interact with mammary epithelial cells during regeneration of the gland, providing evidence for the ascendancy of the tissue microenvironment over the intrinsic nature of cells from an alternative adult tissue.
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ErbB/EGF Signaling and EMT in Mammary Development and Breast Cancer
TL;DR: The purpose of this review is to summarize current information regarding the role of ErbB signaling in aspects of EMT that influence epithelial cell plasticity during mammary gland development and tumorigenesis.
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The mammary microenvironment alters the differentiation repertoire of neural stem cells
Brian W. Booth,David L. Mack,Andreas Androutsellis-Theotokis,Ronald D.G. McKay,Corinne A. Boulanger,Gilbert H. Smith +5 more
TL;DR: The conclusion that tissue-specific signals emanating from the stroma and from the differentiated somatic cells of the mouse mammary gland can redirect the NSCs to produce cellular progeny committed to MEC fates is supported.
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Estrogen receptor-α and progesterone receptor are expressed in label-retaining mammary epithelial cells that divide asymmetrically and retain their template DNA strands
Brian W. Booth,Gilbert H. Smith +1 more
TL;DR: The results presented here support the premise that there is a subpopulation of LRECs in the murine mammary gland that is positive for ER-α and/or PR, raising the possibility that LREC comprise a hierarchy of asymmetrically cycling mammary stem/progenitor cells that are distinguished by the presence or absence of nuclear steroid receptor expression.
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Interleukin-13 Induces Proliferation of Human Airway Epithelial CellsIn Vitro via a Mechanism Mediated by Transforming Growth Factor- α
TL;DR: This study provides the first experimental evidence that IL-13 can initiate a proliferative response of human airway epithelium in the absence of inflammatory cells or other cell types.