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Bruce M. Hendry
Researcher at King's College London
Publications - 132
Citations - 4445
Bruce M. Hendry is an academic researcher from King's College London. The author has contributed to research in topics: Kidney & Kidney disease. The author has an hindex of 35, co-authored 132 publications receiving 4040 citations. Previous affiliations of Bruce M. Hendry include St Helier Hospital & Tel Aviv University.
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Journal ArticleDOI
Tenofovir-associated kidney toxicity in HIV-infected patients: a review of the evidence.
TL;DR: The nature and extent of TDF-associated kidneyoxicity in the HIV-infected population, potential underlying mechanisms of toxicity in the proximal tubule, and risk factors for developing tubular dysfunction are discussed and suggested strategies to monitor patients on TDF therapy are suggested.
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The differential role of Smad2 and Smad3 in the regulation of pro-fibrotic TGFβ1 responses in human proximal-tubule epithelial cells
TL;DR: Different roles for Smad2 and Smad3 are demonstrated in TGFbeta1-induced CTGF expression and markers of EMT in human PTECs, which can be of therapeutic value in designing targeted anti-fibrotic therapies for tubulo-interstitial fibrosis.
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A randomized multicenter trial of the anti-ICAM-1 monoclonal antibody (enlimomab) for the prevention of acute rejection and delayed onset of graft function in cadaveric renal transplantation: a report of the European Anti-ICAM-1 Renal Transplant Study Group.
Kaija Salmela,L. Wramner,Henrik Ekberg,Ingeborg A. Hauser,Øystein Bentdal,Lars-eric Lins,Helena Isoniemi,Lars Bäckman,Persson Nh,Hans-Hellmut Neumayer,Päl Foyn Jørgensen,Claus Spieker,Bruce M. Hendry,Anthony Nicholls,Günter Kirste,Georg Hasche +15 more
TL;DR: Short term enlimomab induction therapy after renal transplantation did not reduce the rate of acute rejection or DGF and there was no statistically significant difference between the incidences between the two groups.
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The molecular mechanisms of anaesthesia
TL;DR: New evidence is presented concerning the anaesthetic properties of the n-alkanes which points to the bilayer as the site of action and also suggests a mechanism for the inhibition of impulse propagation.
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Transplant renal artery stenosis in 77 patients--does it have an immunological cause?
Wilson Wong,Simon P. Fynn,Robert Higgins,Hugh Walters,Sarah Evans,Colin Deane,David Goss,M. Bewick,S. Snowden,John E. Scoble,Bruce M. Hendry +10 more
TL;DR: Recurrence but not occurrence of TRAS was associated with the use of cyclosporine, and there was a significantly higher incidence of rejection, especially cellular rejection in the TRAS group.