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Bruno Crestani
Researcher at French Institute of Health and Medical Research
Publications - 393
Citations - 14981
Bruno Crestani is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Medicine & Idiopathic pulmonary fibrosis. The author has an hindex of 55, co-authored 293 publications receiving 11785 citations. Previous affiliations of Bruno Crestani include McMaster University & Pasteur Institute.
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Journal ArticleDOI
An Official American Thoracic Society/European Respiratory Society Statement: Update of the International Multidisciplinary Classification of the Idiopathic Interstitial Pneumonias
William D. Travis,Ulrich Costabel,David M Hansell,Talmadge E. King,David A. Lynch,Andrew G. Nicholson,Christopher J. Ryerson,Jay H. Ryu,Moisés Selman,Athol U. Wells,Jurgen Behr,Demosthenes Bouros,Kevin K. Brown,Thomas V. Colby,Harold R. Collard,Carlos Robalo Cordeiro,Vincent Cottin,Bruno Crestani,Marjolein Drent,Rosalind F. Dudden,Jim Egan,Kevin R. Flaherty,Cory M. Hogaboam,Yoshikazu Inoue,Takeshi Johkoh,Dong Soon Kim,M Kitaichi,James E. Loyd,Fernando J. Martinez,Jeffrey L. Myers,Shandra Protzko,Ganesh Raghu,Luca Richeldi,Nicola Sverzellati,Jeffrey J. Swigris,Dominique Valeyre +35 more
TL;DR: This update is a supplement to the previous 2002 IIP classification document and outlines advances in the past decade and potential areas for future investigation.
Journal ArticleDOI
EULAR/ERA-EDTA recommendations for the management of ANCA-associated vasculitis.
Max Yates,Max Yates,Richard A. Watts,Ingeborg M. Bajema,Maria C. Cid,Bruno Crestani,Thomas H. Hauser,Bernhard Hellmich,Julia U Holle,Martin Laudien,Mark A. Little,Raashid Luqmani,Alfred Mahr,Peter A. Merkel,John Mills,Janice Mooney,Mårten Segelmark,Vladimir Tesar,Kerstin Westman,Augusto Vaglio,Nilüfer Yalçındağ,David Jayne,Chetan Mukhtyar +22 more
TL;DR: The 2009 European League Against Rheumatism recommendations for the management of antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis (AAV) have been updated and 15 recommendations were developed, covering general aspects, such as attaining remission.
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Extracellular ATP is a danger signal activating P2X7 receptor in lung inflammation and fibrosis.
Nicolas Riteau,Paméla Gasse,Louis Fauconnier,Aurélie Gombault,Marion Couegnat,Lizette Fick,Jean Kanellopoulos,Valérie F. J. Quesniaux,Sylvain Marchand-Adam,Bruno Crestani,Bernhard Ryffel,Isabelle Couillin +11 more
TL;DR: The role of extracellular adenosine triphosphate (eATP) in pulmonary inflammation and fibrosis is addressed and ATP released from BLM-injured lung cells constitutes a major endogenous danger signal that engages the P2X(7) receptor/pannexin-1 axis, leading to IL-1β maturation and lung fibrosis.
Journal ArticleDOI
Idiopathic Pulmonary Fibrosis (an Update) and Progressive Pulmonary Fibrosis in Adults: An Official ATS/ERS/JRS/ALAT Clinical Practice Guideline
Ganesh Raghu,Martine Remy-Jardin,Luca Richeldi,Carey C. Thomson,Yoshikazu Inoue,Takeshi Johkoh,Michael Kreuter,David A. Lynch,Toby M. Maher,Fernando J. Martinez,Maria Molina-Molina,Jeffrey L. Myers,Andrew G. Nicholson,Christopher J. Ryerson,Mary E. Strek,Lauren K. Troy,Marlies S. Wijsenbeek,Manoj J. Mammen,Tanzib Hossain,Brittany D Bissell,Derrick D. Herman,Stephanie Hon,Fayez Kheir,Yet H. Khor,Madalina Macrea,Katerina M. Antoniou,D. Bouros,Ivette Buendía-Roldán,Fabian Caro,Bruno Crestani,Lawrence A. Ho,Julie Morisset,Amy L. Olson,Anna J. Podolanczuk,Venerino Poletti,Moisés Selman,Thomas J Ewing,Stephen Jones,Shandra L Knight,Marya Ghazipura,Kevin C. Wilson +40 more
TL;DR: A conditional recommendation was made to regard transbronchial lung cryobiopsy as an acceptable alternative to surgical lung biopsy in centers with appropriate expertise and additional research into pirfenidone was recommended.
Journal ArticleDOI
NOX4/NADPH oxidase expression is increased in pulmonary fibroblasts from patients with idiopathic pulmonary fibrosis and mediates TGFbeta1-induced fibroblast differentiation into myofibroblasts.
TL;DR: NOX4 is critical for modulation of the pulmonary myofibroblast phenotype in IPF, probably by modulating the response to TGFβ1 and PDGF.