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Bruno M. Moerschbacher

Researcher at University of Münster

Publications -  182
Citations -  5934

Bruno M. Moerschbacher is an academic researcher from University of Münster. The author has contributed to research in topics: Chitin & Chitosan. The author has an hindex of 37, co-authored 170 publications receiving 4874 citations. Previous affiliations of Bruno M. Moerschbacher include RWTH Aachen University & Oklahoma State University–Stillwater.

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Comparison of the Ability of Partially N-acetylated Chitosans and Chitooligosaccharides to Elicit Resistance Reactions in Wheat Leaves

TL;DR: The results suggest that different mechanisms are involved in the elicitation of POD activities by GlcNAc oligomers, and of PAL and Pod activities by partially N-acetylated chitosan polymers and that both enzymes have to be activated for lignin biosynthesis and ensuing necrosis to occur.
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Developmentally regulated conversion of surface‐exposed chitin to chitosan in cell walls of plant pathogenic fungi

TL;DR: Surface modification of the fungal cell walls by chitin de- N -acetylation is discussed as a fungal strategy to protect cell walls of pathogenic hyphae from enzymatic hydrolysis by host chit inases, and to avoid generation of an auto-catalytic defense response system in the invaded host tissue.
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Parameters influencing the size of chitosan-TPP nano- and microparticles

TL;DR: By controlling two simple factors of the polymer solution, namely its initial concentration and its solvent environment, it is feasible to control in a reproducible manner the production and characteristics of chitosan particles ranging in size from nano- to micrometres.
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Complexation of copper(II) with chitosan nanogels: Toward control of microbial growth

TL;DR: The suitability of these copper(II)-chitosan colloids as a new generation of copper-based bio-pesticides, i.e. as a bio-compatible, bio-active and pH-sensitive delivery system, is discussed.
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Specific Inhibition of Lignification Breaks Hypersensitive Resistance of Wheat to Stem Rust

TL;DR: Treatment with any of the cinnamyl-alcohol dehydrogenase inhibitors decreased the frequency of lignified necrotic host cells and concomitantly led to increased fungal growth, pointing to a causal relationship between the formation of lIGNin precursors and the resistance of wheat to stem rust.