C
Carlo Alberto Tassinari
Researcher at University of Bologna
Publications - 274
Citations - 13053
Carlo Alberto Tassinari is an academic researcher from University of Bologna. The author has contributed to research in topics: Epilepsy & Ictal. The author has an hindex of 61, co-authored 273 publications receiving 12435 citations. Previous affiliations of Carlo Alberto Tassinari include University of Wisconsin-Madison & University of Turku.
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Journal ArticleDOI
Transcranial magnetic stimulation in epileptic patients: usefulness and safety.
Carlo Alberto Tassinari,Roberto Michelucci,A. Forti,Rosaria Plasmati,Walter Troni,Fabrizio Salvi,M. Blanco,Guido Rubboli +7 more
TL;DR: TMS, as currently used for monitoring conduction in central motor pathways, does not induce seizures in drug-treated epileptic patients.
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Therapy of encephalopathy with status epilepticus during sleep (ESES/CSWS syndrome): an update
Pierangelo Veggiotti,Maria Carmela Pera,Federica Teutonico,Daniela Brazzo,Umberto Balottin,Carlo Alberto Tassinari +5 more
TL;DR: Treatment options for ESES/CSWS include some antiepileptic drugs, steroids, immunoglobulins, the ketogenic diet, and surgery (multiple subpial transections); the comparative value of each of these treatments is reviewed and a personal therapeutic approach is proposed.
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Lafora disease due to EPM2B mutations: a clinical and genetic study.
C. Gómez-Abad,Pilar Gómez-Garre,Eva Gutierrez-Delicado,Serap Saygi,Roberto Michelucci,Carlo Alberto Tassinari,S. Rodríguez de Córdoba,José M. Serratosa +7 more
TL;DR: Genetic allelic heterogeneity is present in Lafora disease associated with mutations in EPM2B, although patients with E PM2B-associated La fora disease seem to have a slightly milder clinical course.
Journal ArticleDOI
Epileptic negative myoclonus
Renzo Guerrini,C. Dravet,P. Genton,Michelle Bureau,J. Roger,Guido Rubboli,Carlo Alberto Tassinari +6 more
TL;DR: Five patients with partial epilepsy of diverse etiology insidiously developed action-activated jerks, one of which was medically resistant, and in two of them it was continuous and could be defined as epilepsia partialis continua.
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SCARB2 mutations in progressive myoclonus epilepsy (PME) without renal failure
Leanne M. Dibbens,Roberto Michelucci,Antonio Gambardella,F. Andermann,F. Andermann,Guido Rubboli,Marta A. Bayly,Tarja Joensuu,Danya F. Vears,Silvana Franceschetti,Laura Canafoglia,Robyn H. Wallace,Alexander G. Bassuk,David A. Power,Carlo Alberto Tassinari,Eva Andermann,Eva Andermann,Anna-Elina Lehesjoki,Samuel F. Berkovic +18 more
TL;DR: This work hypothesized that mutations in SCARB2 might account for unsolved cases of progressive myoclonus epilepsy (PME) without renal impairment, especially those resembling Unverricht‐Lundborg disease (ULD).