C
Catherine A. Royer
Researcher at Rensselaer Polytechnic Institute
Publications - 241
Citations - 10432
Catherine A. Royer is an academic researcher from Rensselaer Polytechnic Institute. The author has contributed to research in topics: Protein folding & Hydrostatic pressure. The author has an hindex of 54, co-authored 224 publications receiving 9725 citations. Previous affiliations of Catherine A. Royer include University of Paris & Massachusetts Institute of Technology.
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Probing protein folding and conformational transitions with fluorescence.
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Revisiting volume changes in pressure-induced protein unfolding
TL;DR: There is evidence that, due to differences in the thermal expansivity of the folded and unfolded states of proteins reported in a half dozen manuscripts, that the sign of the volume change may become positive at higher temperatures.
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Pressure provides new insights into protein folding, dynamics and structure.
TL;DR: Kinetic studies under pressure enable dissection of the roles of packing and cavities in folding, and in assembly of multimolecular structures such as protein-DNA complexes and viruses.
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Cavities determine the pressure unfolding of proteins
Julien Roche,Jose A. Caro,Douglas R. Norberto,Philippe Barthe,Christian Roumestand,Jamie L. Schlessman,Angel E. Garcia,E Bertrand García-Moreno,Catherine A. Royer +8 more
TL;DR: The promise of pressure perturbation is illustrated as a unique tool for examining the roles of packing, conformational fluctuations, and water penetration as determinants of solution properties of proteins, and for detecting folding intermediates and other structural details of protein-folding landscapes that are invisible to standard experimental approaches.
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Scavenger receptor class B type I is a key host factor for hepatitis C virus infection required for an entry step closely linked to CD81
Mirjam B. Zeisel,George Koutsoudakis,E.K. Schnober,A. Haberstroh,A. Haberstroh,Hubert E. Blum,François-Loïc Cosset,François-Loïc Cosset,Takaji Wakita,Daniel Jaeck,Michel Doffoel,Catherine A. Royer,Eric Soulier,Evelyne Schvoerer,Catherine Schuster,Françoise Stoll-Keller,Ralf Bartenschlager,Thomas Pietschmann,Heidi Barth,Thomas F. Baumert +19 more
TL;DR: The data suggest that SR‐BI represents a key host factor forHCV entry, is implicated in the same HCV entry pathway as CD81, and targets an entry step closely linked to HCV–CD81 interaction.