C
Cedric S. Raine
Researcher at Albert Einstein College of Medicine
Publications - 296
Citations - 27558
Cedric S. Raine is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Myelin & Multiple sclerosis. The author has an hindex of 82, co-authored 296 publications receiving 26593 citations. Previous affiliations of Cedric S. Raine include Yeshiva University & Icahn School of Medicine at Mount Sinai.
Papers
More filters
Journal ArticleDOI
Transformation of cells of astrocyte lineage into macrophage-like cells in organotypic cultures of mouse spinal cord tissue.
TL;DR: It thus appears that immature neuroepithelial cells of astrocytic lineage are capable of transforming into macrophage-like cells in organotypic culture.
Journal ArticleDOI
Multiple sclerosis: death receptor expression and oligodendrocyte apoptosis in established lesions
TL;DR: It is postulate that while oligodendrocyte apoptosis might prevail during the initial stages of MS, from the findings other mechanisms probably account for their loss in the established lesion and decoy receptors may play a protective role in oligodendedrocytes survival.
Journal ArticleDOI
Chronic relapsing experimental allergic encephalomyelitis ☆: Correlation of circulating lymphocyte fluctuations with disease activity in suppressed and unsuppressed animals
TL;DR: The results indicate that chronic relapsing EAE can be successfully suppressed with MBP in IFA, that this suppression is permanent and that the immunologic findings presented correlate well with the clinical and pathologic facets of the disease.
Journal ArticleDOI
Membrane specialisations between demyelinated axons and astroglia in chronic EAE lesions and multiple sclerosis plaques.
TL;DR: In this paper, the authors describe membrane specialisations between naked CNS axons and fibrous astroglial cell processes, developing subsequent to demyelination, which may have physiological implications.
Journal ArticleDOI
Antisera to different glycolipids induce myelin alterations in mouse spinal cord tissue cultures.
TL;DR: In addition to anti-galactocerebroside activity, which was previously found to be the major antibody responsible for the demyelinating activity induced by anti-whole CNS tissue antiserum, data suggest that antibodies to gangliosides like GM1 and GM4 might also play a role in immune-mediated demyELination, including perhaps, the human demyelininating diseases.