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Cedric S. Raine
Researcher at Albert Einstein College of Medicine
Publications - 296
Citations - 27558
Cedric S. Raine is an academic researcher from Albert Einstein College of Medicine. The author has contributed to research in topics: Myelin & Multiple sclerosis. The author has an hindex of 82, co-authored 296 publications receiving 26593 citations. Previous affiliations of Cedric S. Raine include Yeshiva University & Icahn School of Medicine at Mount Sinai.
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The long-term effects of a single injection of taxol upon peripheral nerve axons.
TL;DR: The cytoskeletal reorganization described supports the concept of the differential movement of axoplasmic neurofilament and that neurofilaments stabilize axonal structures and shows taxol to have a long-lasting and local effect upon axoplasmsic organizationin vivo.
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Transcellular migration of neutrophils is a quantitatively significant pathway across dermal microvascular endothelial cells
TL;DR: This in vitro model closely simulates the physiological process of neutrophil extravasation in vivo and can be further utilized to evaluate the relative contribution of distinct migratory pathways to the pathophysiology of inflammatory skin disease.
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Multiple sclerosis: Heterology among early T cells and TG cells
TL;DR: It was found that TG cells are a T cell subpopulation distinct from early T cells, and by examining the distribution of TG cells in suspensions of isolated early and isolated late T cells.
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Axoplasmic asymmetry at the node of Ranvier
TL;DR: The previously described unilateral condensation of axoplasmic organelles at the node of Ranvier of large diameter fibres in spinal nerve roots has been confirmed in a single normal rabbit.
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Structural dynamics of oligodendrocyte lysis by perforin in culture: relevance to multiple sclerosis.
TL;DR: A successful method to culture SJL mouse oligodendrocytes is reported and lysis of these cells in vitro mediated by the pore‐forming protein, perforin, a candidate effector molecule in inflammatory demyelination is analyzed, leaving us to conclude that per forin may play an important role in the human disease.