C
Chantal Kemner
Researcher at Utrecht University
Publications - 165
Citations - 9769
Chantal Kemner is an academic researcher from Utrecht University. The author has contributed to research in topics: Autism & Pervasive developmental disorder. The author has an hindex of 50, co-authored 162 publications receiving 9040 citations. Previous affiliations of Chantal Kemner include Maastricht University & University of Michigan.
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Keep your eyes on development: the behavioral and neurophysiological development of visual mechanisms underlying form processing.
TL;DR: An overview of the current understanding of the typical development, from birth to adulthood, of form-characteristic processing, as measured both behaviorally and neurophysiologically is provided.
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Parallel development of ERP and behavioural measurements of visual segmentation.
TL;DR: The results reveal that visual segmentation continues to develop until early puberty, and only by 13-14 years of age, children do integrate and segregate visual information as adults do.
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Sensory gating and sensorimotor gating in medication-free obsessive-compulsive disorder patients.
TL;DR: It was concluded that sensorimotor and sensory gating is not impaired in drug-free OCD patients, taking into account the menstrual cycle effects in women, and hypotheses linking deficits in these inhibition paradigms and the pathogenesis of OCD are not supported.
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ERPs and Eye Movements Reflect Atypical Visual Perception in Pervasive Developmental Disorder
TL;DR: It is suggested that subjects with PDD show abnormal activation of visual pathways dedicated to the processing of high and low spatial frequencies.
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Haloperidol counteracts the ketamine-induced disruption of processing negativity, but not that of the P300 amplitude.
Bob Oranje,Christine C. Gispen-de Wied,Herman G.M. Westenberg,Chantal Kemner,Marinus N. Verbaten,René S. Kahn +5 more
TL;DR: The current results suggest that ketamine reduced P300 amplitude by its antagonistic effect on glutamatergic activity, while it reduced processing negativity by its agonists effect on dopaminergic D2 activity.