C
Charles van Ypersele de Strihou
Researcher at Université catholique de Louvain
Publications - 115
Citations - 6244
Charles van Ypersele de Strihou is an academic researcher from Université catholique de Louvain. The author has contributed to research in topics: Transplantation & Glycation. The author has an hindex of 46, co-authored 115 publications receiving 6065 citations. Previous affiliations of Charles van Ypersele de Strihou include Tokai University & Nagoya University.
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Journal ArticleDOI
Implication of an increased oxidative stress in the formation of advanced glycation end products in patients with end-stage renal failure
Toshio Miyata,Yoshinao Wada,Zhe Cai,Yoshiyasu Iida,Katsunori Horie,Yoshinari Yasuda,Kenji Maeda,Kiyoshi Kurokawa,Charles van Ypersele de Strihou +8 more
TL;DR: It is shown that, in uremia, CML and pentosidine production is determined both by an increased oxidative stress and the availability of precursors such as oxiAA, which suggests that AGE, that is, C ML andpentosidine, production is accelerated under oxidative stress, even in the absence of glucose.
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Angiotensin II Receptor Antagonists and Angiotensin-Converting Enzyme Inhibitors Lower In Vitro the Formation of Advanced Glycation End Products: Biochemical Mechanisms
Toshio Miyata,Charles van Ypersele de Strihou,Yasuhiko Ueda,Kohji Ichimori,Reiko Inagi,Hiroshi Onogi,Naoyoshi Ishikawa,Masaomi Nangaku,Kiyoshi Kurokawa +8 more
TL;DR: It is demonstrated for the first time that widely used hypotensive agents, AIIR antagonists and ACE inhibitors, significantly attenuate AGE production.
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Urothelial lesions in Chinese-herb nephropathy
Jean-Pierre Cosyns,Michel Jadoul,Jean-Paul Squifflet,François-Xavier Wese,Charles van Ypersele de Strihou +4 more
TL;DR: Results show that the intake of Chinese herbs containing AA has a dramatic carcinogenic effect and is associated with the overexpression of p53, which suggests a role for a p53 gene mutation.
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Chinese herbs nephropathy: A clue to Balkan endemic nephropathy?
Jean-Pierre Cosyns,Jean-Pierre Cosyns,Michel Jadoul,Michel Jadoul,Jean-Paul Squifflet,Jean-Paul Squifflet,Jean-François De Plaen,Jean-François De Plaen,Dusan Ferluga,Dusan Ferluga,Charles van Ypersele de Strihou,Charles van Ypersele de Strihou +11 more
TL;DR: On morphological and clinical grounds, CHN appears similar to BEN; a common etiologic agent, aristolochic acid, is suspected and the known carcinogenic potential of this compound is taken together with the finding of multiple foci of cellular atypia of the urothelium suggest that CHN patients should undergo a regular follow-up for Urothelial malignancy.
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Advanced glycation and lipoxidation end products: Role of reactive carbonyl compounds generated during carbohydrate and lipid metabolism
TL;DR: The uremic syndrome is usually attributed to the retention of a variety of compounds as a result of a deficient renal clearance, but as recently noted by Vanholder and De Smet ([1][1]), this is not the case.