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Christophe Girard

Researcher at French Institute of Health and Medical Research

Publications -  39
Citations -  4712

Christophe Girard is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Kir6.2 & Melanoma. The author has an hindex of 25, co-authored 37 publications receiving 4263 citations. Previous affiliations of Christophe Girard include Centre national de la recherche scientifique & University of Oxford.

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Overexpression of Fto leads to increased food intake and results in obesity.

TL;DR: This study provides the first direct evidence that increased Fto expression causes obesity in mice and shows here that ubiquitous overexpression of Fto leads to a dose-dependent increase in body and fat mass, irrespective of whether mice are fed a standard or a high-fat diet.
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Control of Pancreatic β Cell Regeneration by Glucose Metabolism

TL;DR: Intracellularly, genetic and pharmacologic manipulations reveal that glucose induces β cell replication via metabolism by glucokinase, the first step of glycolysis, followed by closure of K(ATP) channels and membrane depolarization, providing a molecular mechanism for homeostatic control of β cell mass by metabolic demand.
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Permanent neonatal diabetes caused by dominant, recessive, or compound heterozygous SUR1 mutations with opposite functional effects.

TL;DR: A novel mutational mechanism was observed in which a heterozygous activating mutation resulted in PNDM only when a second, loss-of-function mutation was also present, and functional studies of selected mutations showed a reduced response to ATP consistent with an activating mutation that results in reduced insulin secretion.
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A heterozygous activating mutation in the sulphonylurea receptor SUR1 (ABCC8) causes neonatal diabetes

TL;DR: It is demonstrated that SUR1 mutations constitute a new genetic aetiology for neonatal diabetes and that they act by reducing the K(ATP) channel's ATP sensitivity.