C
Christopher A. Jolly
Researcher at University of Texas at Austin
Publications - 31
Citations - 733
Christopher A. Jolly is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Inflammation & Acyltransferase. The author has an hindex of 13, co-authored 27 publications receiving 668 citations.
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A resilience intervention in African American adults with type 2 diabetes: a pilot study of efficacy.
TL;DR: The pilot study documented the feasibility and potential effectiveness of the Diabetes Coaching Program to enhance diabetes empowerment, diabetes self-management, and reductions in the progression of obesity, type 2 diabetes, and cardiovascular disease in the African American community.
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Dietary Restriction and Immune Function
TL;DR: Understanding the biochemical and molecular targets of dietary restriction in the immune system may lead to the development of new dietary strategies to delay or prevent the onset of aging, cancer, and autoimmune disease.
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NSAID Use Reduces Breast Cancer Recurrence in Overweight and Obese Women: Role of Prostaglandin–Aromatase Interactions
Laura W. Bowers,Ilane X.F. Maximo,Andrew Brenner,Muralidhar Beeram,Stephen D. Hursting,Ramona S. Price,Rajeshwar Rao Tekmal,Christopher A. Jolly,Linda A. deGraffenried +8 more
TL;DR: It is demonstrated that daily use of a nonsteroidal anti-inflammatory drug (NSAID), which inhibits COX-2 activity, is associated with reduced estrogen receptor α (ERα)-positive breast cancer recurrence in obese and overweight women.
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n−3 Polyunsaturated fatty acids exert immunomodulatory effects on lymphocytes by targeting plasma membrane molecular organization
TL;DR: Recent data indicating n-3 PUFAs exert immunosuppressive effects on the function of effector and regulatory CD4(+) T cells are focused on and new functional and mechanistic questions are proposed that will aid in the development of fish oil as adjuvant therapy for treating unresolved chronic inflammation.
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Dietary α-linolenic acid increases brain but not heart and liver docosahexaenoic acid levels
TL;DR: It is demonstrated that 18∶3n−3-enriched diets reduced tissue 20∶4n−6 levels and increased cellular n−3 levels in a tissuedependent manner, thereby presenting tissue with this EFA for further elongation and desaturation by the brain.