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Clemens Kirschbaum

Researcher at Dresden University of Technology

Publications -  519
Citations -  67877

Clemens Kirschbaum is an academic researcher from Dresden University of Technology. The author has contributed to research in topics: Trier social stress test & Cortisol secretion. The author has an hindex of 117, co-authored 488 publications receiving 61570 citations. Previous affiliations of Clemens Kirschbaum include University of Düsseldorf & University of Trier.

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Acute stress responses in salivary alpha-amylase predict increases of plasma norepinephrine

TL;DR: Examination of relation between stress-induced sAA activity with NE and E by exposing a larger and less constrained sample to an effective stress protocol shows an association between stress responses of sAA and plasma NE, which is stronger than the association of norepinephrine and epinephrine responses, thus placing the predictive power of s AA well within the expected range for different SNS markers.
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Heritability of cortisol responses to human corticotropin-releasing hormone, ergometry, and psychological stress in humans.

TL;DR: Cortisol changes after bicycle ergometry revealed no impact of genetic factors on the secretion of cortisol in response to strenuous physical exercise, and heredity appeared to be play a minor role in the adrenocortical response to psychological stress.
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Impact of Antenatal Synthetic Glucocorticoid Exposure on Endocrine Stress Reactivity in Term-Born Children

TL;DR: The present study provides the first evidence for long-lasting effects of antenatal synthetic GC exposure on HPA-axis reactivity in term-born children, and may bear important implications regarding the vulnerability for stress-related physical and psychiatric disorders.
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Inflexibly focused under stress: Acute psychosocial stress increases shielding of action goals at the expense of reduced cognitive flexibility with increasing time lag to the stressor

TL;DR: Evidence is provided for a stress-induced time-dependent decrease of cognitive flexibility that might be related to changes in cortisol levels, which paralleled the time course of the hypothalamus–pituitary–adrenal (HPA) stress response.
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Does cellular aging relate to patterns of allostasis? An examination of basal and stress reactive HPA axis activity and telomere length

TL;DR: Greater cortisol responses to an acute stressor and/or dysregulated patterns of daily cortisol secretion are associated with shorter telomere length, as were higher overnight urinary free cortisol levels and flatter daytime cortisol slopes.