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Darryl L. Russell

Researcher at University of Adelaide

Publications -  112
Citations -  8570

Darryl L. Russell is an academic researcher from University of Adelaide. The author has contributed to research in topics: Ovulation & Oocyte. The author has an hindex of 45, co-authored 108 publications receiving 7641 citations. Previous affiliations of Darryl L. Russell include Prince Henry's Institute of Medical Research & Baylor College of Medicine.

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Progesterone-regulated genes in the ovulation process: ADAMTS-1 and cathepsin L proteases

TL;DR: The identification of two regulated proteases in the ovary, together with their abnormal expression in anovulatory PR knockout mice, suggests that each plays a critical role in follicular rupture and represents a major advance in the understanding of the proteolytic events that control ovulation.
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Ovulation: new dimensions and new regulators of the inflammatory-like response.

TL;DR: This review focuses on recent endocrine, biochemical, and genetic information that has been derived largely from the identification of new genes that are expressed in the ovary, and from knowledge gained by the targeted deletion of genes that appear to impact the ovulation process.
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Novel signaling pathways that control ovarian follicular development, ovulation, and luteinization.

TL;DR: This review summarizes some new aspects of peptide and steroid hormone signaling in the rodent ovary that appear to regulate cumulus expansion and other aspects relating to ovarian embryogenesis and possibly ovulation and luteinization.
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Molecular mechanisms of ovulation: co-ordination through the cumulus complex

TL;DR: This review focuses on the recent advances in understanding of molecular mechanisms that commence after the gonadotrophin surge and culminate with release of the oocyte.
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Beta-Oxidation Is Essential for Mouse Oocyte Developmental Competence and Early Embryo Development

TL;DR: The importance of lipid metabolism for oocyte developmental competence and early embryo development was demonstrated by assessing the rate of embryo development following inhibition or upregulation of beta-oxidation with etomoxir (an inhibitor of CPT1B) or l-carnitine, respectively.