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David C. Reutens

Researcher at University of Queensland

Publications -  367
Citations -  11854

David C. Reutens is an academic researcher from University of Queensland. The author has contributed to research in topics: Epilepsy & Cognition. The author has an hindex of 55, co-authored 356 publications receiving 10668 citations. Previous affiliations of David C. Reutens include Royal Perth Hospital & Royal Melbourne Hospital.

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Apoptotic Mechanisms After Cerebral Ischemia

TL;DR: Accumulating evidence now indicates that apoptosis is prevalent in nonneuronal cells and that caspase-independent mechanisms also play a key role in the development of apoptosis in ischemic lesions.
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Research Review: Williams syndrome: a critical review of the cognitive, behavioral, and neuroanatomical phenotype.

TL;DR: The need for methodologically sound studies that utilize multiple comparison groups, developmental trajectories, and longitudinal analyses to examine the WS phenotype is highlighted, as well as those that link brain structure and function to the cognitive and behavioral phenotype of WS individuals.
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Regional Cerebral Blood Flow Changes as a Function of Delta and Spindle Activity during Slow Wave Sleep in Humans

TL;DR: Changes in regional cerebral blood flow in humans during the progression from relaxed wakefulness through slow wave sleep (SWS) were examined as a function of spindle and δ electroencephalographic activity of SWS.
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PK11195 binding to the peripheral benzodiazepine receptor as a marker of microglia activation in multiple sclerosis and experimental autoimmune encephalomyelitis.

TL;DR: The results indicate the potential to develop markers suitable for both in vitro and in vivo use, which will serve to help correlate phenotypic and functional properties of cells which participate in disease or injury responses within the CNS.
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Cerebral White Matter Lesions, Gait, and the Risk of Incident Falls. A Prospective Population-Based Study

TL;DR: These data provide the first prospective evidence to the authors' knowledge demonstrating that WMLs are strong risk factors for falls in the general older population and present potential therapeutic targets for interventional trials in falls prevention.