D
David C. Reutens
Researcher at University of Queensland
Publications - 367
Citations - 11854
David C. Reutens is an academic researcher from University of Queensland. The author has contributed to research in topics: Epilepsy & Cognition. The author has an hindex of 55, co-authored 356 publications receiving 10668 citations. Previous affiliations of David C. Reutens include Royal Perth Hospital & Royal Melbourne Hospital.
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Journal ArticleDOI
Segmentation of the mouse hippocampal formation in magnetic resonance images
Kay L. Richards,Charles Watson,Rachel F. Buckley,Rachel F. Buckley,Nyoman D. Kurniawan,Zhengyi Yang,Marianne D. Keller,Richard Beare,Richard Beare,Perry F. Bartlett,Gary F. Egan,Gary F. Egan,Graham J. Galloway,George Paxinos,George Paxinos,George Paxinos,Steven Petrou,Steven Petrou,David C. Reutens +18 more
TL;DR: A guide for segmenting over 40 structures in an adult mouse brain using 30 μm isotropic resolution images acquired with a 16.4 T MR imaging system and combined using super-resolution reconstruction is developed.
Journal ArticleDOI
Function and organization in dysgenic cortex: Case report
Mark C. Preul,Richard Leblanc,Fernando Cendes,François Dubeau,David C. Reutens,Roberto Spreafico,Giorgio Battaglia,Massimo Avoli,Pierre Langevin,Douglas L. Arnold,Jean Guy Villemure +10 more
TL;DR: These studies demonstrated compensatory cortical reorganization and showed that large areas of heterotopia and cortical dysplasia in the central area may retain normal motor and sensory function despite strikingly altered cytoarchitectonic organization and neuronal metabolism.
Journal ArticleDOI
Prospective assessment of autism traits in children exposed to antiepileptic drugs during pregnancy.
Amanda G. Wood,Caroline Nadebaum,Vicki Anderson,David C. Reutens,Sarah Barton,Terence J. O'Brien,Frank J.E. Vajda +6 more
TL;DR: A prospective cohort study in children exposed to anticonvulsants during pregnancy, with all assessments conducted by examiners who were blinded to drug‐exposure status is presented.
Journal ArticleDOI
Cross-ethnic meta-analysis identifies association of the GPX3-TNIP1 locus with amyotrophic lateral sclerosis
Beben Benyamin,Ji He,Qiongyi Zhao,Jacob Gratten,Fleur C. Garton,Paul Leo,Paul Leo,Zhijun Liu,Marie Mangelsdorf,Ammar Al-Chalabi,Lisa Anderson,Lisa Anderson,Tim J. Butler,Lu Chen,Xiang Ding Chen,Katie Cremin,Katie Cremin,Hong Weng Deng,Matthew S Devine,Janette Edson,Jennifer A. Fifita,Sarah Furlong,Ying Ying Han,Jessica Harris,Jessica Harris,Anjali K. Henders,Rosalind L. Jeffree,Zi-Bing Jin,Zhongshan Li,Ting Li,Mengmeng Li,Yong Lin,Xiaolu Liu,Mhairi Marshall,Mhairi Marshall,Emily P. McCann,Bryan J. Mowry,Shyuan T. Ngo,Roger Pamphlett,Shu Ran,David C. Reutens,Dominic B. Rowe,Perminder S. Sachdev,Sonia Shah,Sharon Song,Sharon Song,Li Jun Tan,Lu Tang,Leonard H. van den Berg,Wouter van Rheenen,Jan H. Veldink,Robyn H. Wallace,Lawrie Wheeler,Lawrie Wheeler,Kelly L. Williams,Jinyu Wu,Xin Wu,Jian Yang,Weihua Yue,Zong Hong Zhang,Dai Zhang,Peter G. Noakes,Ian P. Blair,Robert D. Henderson,Robert D. Henderson,Pamela A. McCombe,Peter M. Visscher,Huji Xu,Perry F. Bartlett,Matthew A. Brown,Matthew A. Brown,Naomi R. Wray,Dongsheng Fan +72 more
TL;DR: A significant association of rs10463311 spanning GPX3-TNIP1 with ALS is found and GGNBP2 was identified using gene-based analysis and summary statistics-based Mendelian randomization analysis, although further replication is needed to confirm this result.
Journal ArticleDOI
Inhibition of histone deacetylase in utero causes sociability deficits in postnatal mice
Randal X. Moldrich,Gayeshika Leanage,David T. She,Elliot Dolan-Evans,Michael James Nelson,Nargis Reza,David C. Reutens +6 more
TL;DR: The data indicate that embryonic HDAC inhibition alone can cause abnormal social behaviors in mice, which serves as a molecular understanding of infant outcomes following mild VPA exposure in utero.