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David C. Reutens

Researcher at University of Queensland

Publications -  367
Citations -  11854

David C. Reutens is an academic researcher from University of Queensland. The author has contributed to research in topics: Epilepsy & Cognition. The author has an hindex of 55, co-authored 356 publications receiving 10668 citations. Previous affiliations of David C. Reutens include Royal Perth Hospital & Royal Melbourne Hospital.

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Reorganisation of the visual cortex in callosal agenesis and colpocephaly

TL;DR: Using functional magnetic resonance imaging (fMRI) in a patient with callosal agenesis and colpocephaly, whose visual acuity and binocular visual fields were normal, the activation pattern was compared to that seen in six normal volunteers.
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Mouse EEG spike detection based on the adapted continuous wavelet transform

TL;DR: A new algorithm for detecting and classifying interictal spikes in mouse EEG recordings is proposed, based on the adapted continuous wavelet transform (CWT), which could distinguish EEG spikes from other transient waveforms with high degree of sensitivity and specificity.
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Localised neuronal migration disorder and intractable epilepsy: a prenatal vascular aetiology.

TL;DR: A patient is described with intractable complex partial seizures associated with heterotopic grey matter and cerebral hypoplasia confined to the territory of the left posterior cerebral artery, implicating prenatal ischaemia without frank infarction as the aetiology of the malformation.
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Corpus callosum size and shape alterations in adolescent inhalant users.

TL;DR: The morphology of the corpus callosum, the largest white matter bundle in the brain, is investigated in an adolescent sample of inhalant users to represent the early stages of neurobiological damage associated with chronic inhalant misuse.
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Brief Vibrotactile Stimulation Does Not Increase Cortical Oxygen Consumption When Measured by Single Inhalation of Positron Emitting Oxygen

TL;DR: Vibrotactile stimulation of the hand elicits no increase in oxygen consumption commensurate with the increase in blood flow measured in human sensory cortex, and this finding rejects the hypothesis that failure to detect an increase of oxygen consumption could be an artefact caused by limitations of the method used previously.