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David G. Greenhalgh
Researcher at Shriners Hospitals for Children
Publications - 474
Citations - 18828
David G. Greenhalgh is an academic researcher from Shriners Hospitals for Children. The author has contributed to research in topics: Population & Burn injury. The author has an hindex of 62, co-authored 447 publications receiving 16963 citations. Previous affiliations of David G. Greenhalgh include University of Vermont & Mallinckrodt.
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Journal ArticleDOI
Benefits of early tracheostomy in severely burned children.
TL;DR: Early tracheostomy in severely burned children is safe and effective and it provides a secure airway and may result in improvement in ventilator management for these children.
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A comparison of full-thickness versus split-thickness autografts for the coverage of deep palm burns in the very young pediatric patient
TL;DR: Improved function and decreased need for reconstructive procedures when full-thickness skin grafts are used for the treatment of deep palm burns in young pediatric patients are demonstrated.
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Predicting mortality from burns: The need for age-group specific models
Sandra L. Taylor,MaryBeth Lawless,MaryBeth Lawless,Terese Curri,Soman Sen,Soman Sen,David G. Greenhalgh,David G. Greenhalgh,Tina L Palmieri,Tina L Palmieri +9 more
TL;DR: It is hypothesized that age variably impacts mortality after burn and that age-specific models for children, adults, and seniors will more accurately predict mortality than an all-ages model.
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Temperature threshold for burn injury: an oximeter safety study.
David G. Greenhalgh,Mary Beth Lawless,Bradford B. Chew,Willem A. Crone,Michael E. Fein,Tina L Palmieri +5 more
TL;DR: Pulse oximeter probes are safe up to a temperature of 43 degrees C for at least 8 hours in well-perfused skin but above that temperature, there is a risk of burn injury.
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Long term dynamics in a mathematical model of HIV-1 infection with delay in different variants of the basic drug therapy model
TL;DR: In this paper, the authors considered a similar type of approximate model incorporating time delay in the process of infection on the healthy T cells which, in turn, implies inclusion of a similar delay in viral replication.