D
David R. Rubinow
Researcher at University of North Carolina at Chapel Hill
Publications - 375
Citations - 25515
David R. Rubinow is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Mood & Premenstrual dysphoric disorder. The author has an hindex of 82, co-authored 364 publications receiving 23457 citations. Previous affiliations of David R. Rubinow include National Institutes of Health & George Washington University.
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Allopregnanolone as a mediator of affective switching in reproductive mood disorders
TL;DR: The role of the “neuroactive” steroids estradiol and progesterone in reproductive endocrine-related mood disorders are described and potential mechanisms by which ALLO might contribute to their pathophysiology are highlighted.
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Cerebrospinal fluid somatostatin and psychiatric illness
TL;DR: There is indirect supporting evidence for a role for somatostatin dysregulation in the most consistently observed biological abnormality in depression, escape from dexamethasone suppression.
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Effect of Menstrual Cycle Phase on Neuroendocrine and Behavioral Responses to the Serotonin Agonist m-Chlorophenylpiperazine in Women with Premenstrual Syndrome and Controls
TL;DR: In this article, the effects of the menstrual cycle phase on neuroendocrine and behavioral responses to the serotonergic agent m-chlorophenylpiperazine (m-CPP) in women with PMS and controls were investigated.
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Oestrogen Receptor α Localisation in the Prefrontal Cortex of Three Mammalian Species
Deidra M. Montague,Cynthia Shannon Weickert,Cynthia Shannon Weickert,Eva Tomaskovic-Crook,Eva Tomaskovic-Crook,Debora A. Rothmond,Debora A. Rothmond,Joel E. Kleinman,David R. Rubinow +8 more
TL;DR: Evidence is provided that ER α protein exists in neurones in mammalian PFC and that ERα is anatomically well‐positioned to directly mediate oestrogen action in these neurones.
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Sex-Related Differences in Stimulated Hypothalamic- Pituitary-Adrenal Axis during Induced Gonadal Suppression
TL;DR: These data confirm earlier reports of sex differences in stimulated HPA axis activity and demonstrate that these differences exist even under induced hypogonadal conditions (i.e. in the absence of characteristic differences in reproductive steroids).