D
David R. Rubinow
Researcher at University of North Carolina at Chapel Hill
Publications - 375
Citations - 25515
David R. Rubinow is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Mood & Premenstrual dysphoric disorder. The author has an hindex of 82, co-authored 364 publications receiving 23457 citations. Previous affiliations of David R. Rubinow include National Institutes of Health & George Washington University.
Papers
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Conditioning and sensitisation in the longitudinal course of affective illness.
TL;DR: The sensitisation models provide a conceptual approach to previously inexplicable clinical phenomena in the longitudinal course of affective illness and may provide a bridge between psychoanalytic/psychosocial and neurobiological formulations of manic-depressive illness.
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Effects of vasopressin on human memory functions
Herbert Weingartner,Philip H. Gold,James C. Ballenger,Sheila A. Smallberg,Richard Summers,David R. Rubinow,Robert M. Post,Frederick K. Goodwin +7 more
TL;DR: Cognitively unimpaired, as well as cognitively impaired adults, treated with DDAVP for a period of several days, learn information more effectively, as measured by the completeness, organization, and consistency of recall.
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Effects of ovarian hormones on human cortical excitability.
TL;DR: An excitatory neuronal effect associated with estradiol is demonstrated and the earlier finding of inhibition associated with progesterone is confirmed.
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Impaired recognition of affect in facial expression in depressed patients.
David R. Rubinow,Robert M. Post +1 more
TL;DR: Depressed patients were significantly impaired in the recognition of affect in the facial, but not verbal, expressions, and the relevance of the observed perceptual deficit in depressed patients to the pathophysiology and symptomatology of depression is discussed.
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The role of reproductive hormones in postpartum depression.
TL;DR: The hypothesis that fluctuations in reproductive hormone levels during pregnancy and the postpartum period trigger PPD in susceptible women is examined to propose that these women constitute a “hormone-sensitive” PPD phenotype, which should be studied independent of other PPD phenotypes to identify underlying pathophysiology and develop novel treatment targets.