世界経済・社会統計 = World development indicators

计量经济分析 = Econometric analysis

How to Do Things With Words

https://link.springer.com/content/pdf/10.2307%2F3343168.pdf

Development as Freedom

The 2008 crash has left all the established economic doctrines - equilibrium models, real business cycles, disequilibria models - in disarray. Part of the problem is due to Smith’s "veil of ignorance": individuals unknowingly pursue society’s interest and, as a result, have no clue as to the macroeconomic effects of their actions: witness the Keynes and Leontief multipliers, the concept of value added, fiat money, Engel’s law and technical progress, to name but a few of the macrofoundations of microeconomics. A good viewpoint to take bearings anew lies in comparing the post-Great Depression institutions with those emerging from Thatcher and Reagan’s economic policies: deregulation, exogenous vs. endoge- nous money, shadow banking vs. Volcker’s Rule. Very simply, the banks, whose lending determined deposits after Roosevelt, and were a public service became private enterprises whose deposits determine lending. These underlay the great moderation preceding 2006, and the subsequent crash.

The Economic Institutions of Capitalism

Received: October 6th, 2020 ; Accepted: February 1st, 2021 ; Published: March 18th, 2021 ; Correspondence: tonchopenev@abv.bg

Study of influence of heat stress on some physiological and productive traits in Holstein-Friesian dairy cows

Human Relations has long hosted a dialogue between entrepreneurship, work, and organization scholars. Research on topics such as entrepreneurial narratives (Kibler, Mandl, Farny, & Salmivaara, 2021), leadership as entrepreneurial agency (Edwards & Meliou, 2015), entrepreneurial careers (Valette & Culié, 2015) or entrepreneurial identity work (Essers & Benschop, 2009; Essers, Doorewaard, & Benschop, 2013) demonstrates the synergistic potential of these domains by highlighting distinct aspects of human practices in a dynamic world. However, the vast majority of studies of entrepreneurship, work and organization have kept a polite distance by emphasizing domain differences and treating them as externalities (e.g., Storey, Salaman and Platman, 2005; McCabe, 2009). With this Special Issue, we aim to reinvigorate the intersections between entrepreneurship, work, and organization by advancing research on the organizational processes and practices of entrepreneurship at work. To do so, we focus on the notion of “entrepreneuring” (Steyaert, 2007) that puts emphasis on the processes of organization-creation as collective work taking place in new ventures, in existing organizations or between them (Hjorth, 2015). This enables to link two key dimensions of contemporary work: the organizational in entrepreneurship and the entrepreneurial in organizing.

Human Relations special issue call for papers

Our short Dialogue challenges a stream of thought that focuses on drawing what we see as a frivolous contrast between creation and discovery view of entrepreneurship. In the former, entrepreneurs create opportunities ex nihilo; in the latter they discover them as “luggage lost at a train station.” “Fetishism” is a label Roy Suddaby applies to articles that are “detached from the empirical world.” The fundamental question is: “How to make entrepreneurial research more relevant and useful to entrepreneurs?” We advocate computational simulation models with input and outcome parameters docked to empirical studies, with agent action tied to grounded process research.

https://papers.ssrn.com/sol3/Delivery.cfm/SSRN_ID2656397_code1152101.pdf?abstractid=2656397&mirid=1&type=2

Realism, Empiricism, and Fetishism in the Study of Entrepreneurship

Facing the future through entrepreneurship theory: A prospective inquiry framework

Simple Summary Chronic obstructive pulmonary disease (COPD) is characterized by chronic inflammation and oxidative stress, both in the airways and blood, and in other organs. Elevated oxidative stress and inflammation have been reported to affect leucocyte telomere length (LTL). We explored the link between GSTM1 and GSTT1 gene polymorphisms, LTL and COPD risk. For GSTM1 and GSTT1, we genotyped 152 COPD patients and 131 non-affected controls, while for TL, we assessed 91 patients and 88 controls. There was a significant difference in GSTM1 null genotype frequency between the patients and controls (0.59 vs. 0.38, p ≤ 0.000), but such was not found for GSTT1 (p = 0.192). COPD patients carrying the GSTM1 null genotype had shorter telomeres compared to those carrying the non-null genotype (15,720 bp vs. 22,442 bp, p = 0.008); and in controls, the opposite occurred (31,354 bp vs. 17,800 bp, p = 0.020). According to our results GSTM1, but not GSTT1, null genotypes might play role in leucocyte telomere shortening, and thus be involved in the pathogenesis of COPD. Abstract Chronic obstructive pulmonary disease (COPD) is characterized by chronic inflammation and oxidative stress both in the airways and blood and other organs. Elevated oxidative stress and inflammation have been reported to affect leucocyte telomere length (LTL). Glutathione S-transferase (GST) enzymes are a large family of xenobiotic-metabolizing enzymes that utilize different ROS products. We aimed to explore the link between GSTM1 and GSTT1 gene polymorphisms, LTL and COPD risk. For GSTM1, we genotyped 152 COPD patients and 131 non-affected controls; for GSTT1, we genotyped 149 COPD patients and 130 controls. We were able to assess TL for 91 patients and 88 controls. There was a significant difference in the GSTM1 null genotype frequency between the patients and controls (0.59 vs. 0.38, p ≤ 0.000), but such was not found for GSTT1 (p = 0.192). When combining both polymorphisms, we obtained a significantly greater presence of at least one null genotype among patients (0.12 vs. 0.05, p = 0.027). An association between GSTT1 and LTL was not found. COPD patients carrying the GSTM1 null genotype had shorter telomeres compared to those carrying the non-null genotype (15,720 bp vs. 22,442 bp, p = 0.008); as for the controls, it was the opposite (31,354 bp vs. 17,800 bp, p = 0.020). The significance in both groups remained when combining GSTM1 and GSTT1 (COPD (at least one null) 16,409 bp vs. COPD (non-null) 22,092 bp, p = 0.029; control (at least one null) 29,666 bp vs. control (non-null) 16,370 bp, p = 0.027). The total glutathione level in GSTM1 non-null controls was higher compared to the null genotype (15.39 ng/mL vs. 5.53 ng/mL, p = 0.002). In COPD patients, we found no association (p = 0.301). In conclusion, according to our results, GSTM1, but not GSTT1, null genotypes might play a role in leucocyte telomere shortening, and thus be involved in the pathogenesis of COPD.

/pdf/the-leucocyte-telomere-length-gstm1-and-gstt1-null-genotypes-1zx7cyrq.pdf

Dimo Dimov

Papers

Study of influence of heat stress on some physiological and productive traits in Holstein-Friesian dairy cows

Human Relations special issue call for papers

Realism, Empiricism, and Fetishism in the Study of Entrepreneurship

Facing the future through entrepreneurship theory: A prospective inquiry framework

The Leucocyte Telomere Length, GSTM1 and GSTT1 Null Genotypes and the Risk of Chronic Obstructive Pulmonary Disease