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Dong Wan Kim

Researcher at Seoul National University Hospital

Publications -  880
Citations -  61091

Dong Wan Kim is an academic researcher from Seoul National University Hospital. The author has contributed to research in topics: Lung cancer & Crizotinib. The author has an hindex of 89, co-authored 833 publications receiving 49632 citations. Previous affiliations of Dong Wan Kim include University of California, Irvine & Korea University.

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Clinical Course of Neuroendocrine Tumors With Different Origins (the Pancreas, Gastrointestinal Tract, and Lung)

TL;DR: Neuroendocrine tumors from the pancreas, GI tract, and lung showed different clinical characteristics, and stage, grade, and age were prognostic for overall survival (OS) and sex were prognosis for disease-free survival.
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MET exon 14 skipping mutation in triple-negative pulmonary adenocarcinomas and pleomorphic carcinomas: An analysis of intratumoral MET status heterogeneity and clinicopathological characteristics

TL;DR: MET-mutated NSCLC is characterized by older age and by an acinar histology and variable MET expression in patients with adenocarcinoma and pleomorphic carcinomas and the correlation among mutations, protein expression, and gene copy number of MET remain unclear.
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Highly stable sodium storage in 3-D gradational Sb-NiSb-Ni heterostructures

TL;DR: In this paper, a compositionally gradational heterostructure composed of a Ni core, NiSb interface, and attached Sb crystals via pulsed electrodeposition under controlled conditions was realized.
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Pulmonary persistence of graphene nanoplatelets may disturb physiological and immunological homeostasis

TL;DR: It is suggested that the long persistence of GNP in the lung may cause adverse health effects by disturbing immunological‐ and physiological‐homeostasis of the authors' body.
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Preclinical Modeling of Osimertinib for NSCLC With EGFR Exon 20 Insertion Mutations.

TL;DR: Osimertinib is active against EGFR exon 20 insertion-mutant NSCLC and flexibly binds within drug-binding pockets in preclinical models and the IC50 ratios for mutant to wild-type cells were higher than those for third-generation EGFR TKIs.