E
Edward J. Dick
Researcher at Texas Biomedical Research Institute
Publications - 144
Citations - 3218
Edward J. Dick is an academic researcher from Texas Biomedical Research Institute. The author has contributed to research in topics: Baboon & Esophagus. The author has an hindex of 25, co-authored 134 publications receiving 2396 citations. Previous affiliations of Edward J. Dick include Wilford Hall Medical Center.
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Journal ArticleDOI
Trichobezoars in baboons: Trichobezoars in baboons
Diana C. P. Mejido,Edward J. Dick,Priscilla C. Williams,R.M. Sharp,Márcia Cristina Ribeiro Andrade,C.D. DiCarlo,Gene B. Hubbard +6 more
TL;DR: The baboon may present a useful model to evaluate the etiology, genetic predisposition, physiopathology, neurobiology, and treatment response of trichobezoars.
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Parturition in baboons (PAPIO SPP.).
Natalia Schlabritz-Loutsevitch,James Maher,Ryan D. Sullivan,Giancarlo Mari,Mauro Schenone,Harris L. Cohen,R. A. Word,Gene Hubbard,Edward J. Dick +8 more
TL;DR: Information is added from a series of pathological and observational cases to highlight insights and selected complications of birth in Papio spp, based on video-recording of the delivery process, X-ray, MRI, and ultrasound evaluations in pregnant baboons.
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Pleuro-pulmonary endometriosis in baboons (Papio spp.): insights into pathogenesis.
TL;DR: Human pleuro‐pulmonary endometriosis (PPE) is rare and recently, several cases of abdominal endometRIosis in baboons that developed PPE were identified.
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Pauci-immune glomerulonephritis in a captive chimpanzee (Pan troglodytes), and a review of spontaneous cases in animals.
TL;DR: Crescentic glomeruli are the hallmark finding in rapidly progressive glomerulonephritis (RPGN) and are characterized by disruption and proliferation of the glomerular capsule and an influx of cells into Bowman's space.
Journal Article
Further Studies on Barretts Mucosa in Baboons: Metaplastic Glandular Cells Produce Sialomucin
TL;DR: In humans, protracted GER is a disease requiring medication that may lead to BM; AB stains mainly goblet cells and occasional columnar cells in BM; in baboons, BM is a natural postnatal process of adaptation to GER, triggered by regurgitation and rumination.