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Elena Volpi
Researcher at University of Texas Medical Branch
Publications - 185
Citations - 18560
Elena Volpi is an academic researcher from University of Texas Medical Branch. The author has contributed to research in topics: Skeletal muscle & Sarcopenia. The author has an hindex of 65, co-authored 169 publications receiving 16216 citations. Previous affiliations of Elena Volpi include KTU Faculty of Medicine & American Physical Therapy Association.
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Journal ArticleDOI
Muscle Protein Metabolism Responds Similarly to Exogenous Amino Acids in Healthy Younger and Older Adults during NO-Induced Hyperemia
E. Lichar Dillon,Shanon L. Casperson,William J. Durham,Kathleen M. Randolph,Randall J. Urban,Elena Volpi,Masood Ahmad,Michael P. Kinsky,Melinda Sheffield-Moore +8 more
TL;DR: The results suggest that the aging vasculature is responsive to exogenous NO and that there is no age-related difference per se in AA-induced anabolism under such hyperemic conditions.
Journal ArticleDOI
Protein metabolism in women and men: similarities and disparities.
Melissa M. Markofski,Elena Volpi +1 more
TL;DR: Sex differences in protein metabolism are most evident during the main phases of hormonal changes, and may be also due to differences in body composition, but methodological issues and sample size must be considered when designing and evaluating these studies.
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Nutrition and sarcopenia of ageing.
Satoshi Fujita,Elena Volpi +1 more
TL;DR: The most recent findings regarding the role of nutritional factors in the development, prevention and treatment of sarcopenia are reviewed, focusing on the nutritional needs of the elderly; the age-related changes in the response of muscle protein metabolism to feeding and to the endogenous hormones released during feeding.
Journal ArticleDOI
A soy, whey and caseinate blend extends postprandial skeletal muscle protein synthesis in rats
Dustie N Butteiger,Mark B. Cope,P. Liu,Ratna Mukherjea,Elena Volpi,Blake B. Rasmussen,Elaine S Krul +6 more
TL;DR: Single source proteins and protein blends all enhance skeletal MPS after a meal, however, Blend 2 had a delayed FSR peak which was significantly higher than whey protein at 135 min.
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Deficiency in repair of the mitochondrial genome sensitizes proliferating myoblasts to oxidative damage.
Bartosz Szczesny,Gabor Olah,Dillon K. Walker,Dillon K. Walker,Elena Volpi,Blake B. Rasmussen,Csaba Szabó,Sankar Mitra +7 more
TL;DR: It is shown that myoblasts are highly sensitive to ROS-mediated DNA damage, particularly in the mitochondrial genome, due to deficiency in 5’ end processing at the DNA strand breaks, and the process of myoblast differentiation increases mitochondrial biogenesis and the level of total glutathione.