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Eric G. Krause

Researcher at University of Florida

Publications -  106
Citations -  3278

Eric G. Krause is an academic researcher from University of Florida. The author has contributed to research in topics: Hypothalamus & Angiotensin II. The author has an hindex of 33, co-authored 88 publications receiving 2604 citations. Previous affiliations of Eric G. Krause include Johns Hopkins University School of Medicine & Florida State University.

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Pleasurable behaviors reduce stress via brain reward pathways.

TL;DR: It is demonstrated that palatable food intake (limited intake of sucrose drink) reduces neuroendocrine, cardiovascular, and behavioral responses to stress in rats, and it is suggested that the palatable/rewarding properties of Sucrose are necessary and sufficient for stress dampening.
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The renin angiotensin system and the metabolic syndrome.

TL;DR: This review summarizes what is known of the actions of the RAS in the brain and throughout the body to influence various metabolic disorders and special emphasis is given to the role of theRAS in body weight regulation.
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Oxytocin receptors are expressed on dopamine and glutamate neurons in the mouse ventral tegmental area that project to nucleus accumbens and other mesolimbic targets

TL;DR: OTR expression by VTA neurons implicates that OT regulation of reward circuitry is more complex than a direct action on DA neurotransmission.
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Cross talk between AT1 receptors and Toll-like receptor 4 in microglia contributes to angiotensin II-derived ROS production in the hypothalamic paraventricular nucleus

TL;DR: Functional interactions between AT1 receptors and TLR4 in mediating ANG II-dependent microglial activation and oxidative stress within the PVN support a functional interaction between the central renin-angiotensin system and innate immunity in the regulation of neurohumoral outflows from the PVn.
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Reporter mouse strain provides a novel look at angiotensin type-2 receptor distribution in the central nervous system

TL;DR: The use of a bacterial artificial chromosome transgenic AT2R-enhanced green fluorescent protein (eGFP) reporter mouse with recent advances in in situ hybridization (ISH) to circumvent the obstacle of limited ability to effectively localize these receptors at a cellular level in the brain is combined to demonstrate that central AT1R are positioned to regulate blood pressure, metabolism, and stress responses.