F
Fabrizio Salvi
Researcher at University of Bologna
Publications - 221
Citations - 14278
Fabrizio Salvi is an academic researcher from University of Bologna. The author has contributed to research in topics: Transthyretin & Chronic cerebrospinal venous insufficiency. The author has an hindex of 56, co-authored 213 publications receiving 12721 citations. Previous affiliations of Fabrizio Salvi include Academy for Urban School Leadership & University of Ferrara.
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Non-invasive evaluation of the myocardial substrate of cardiac amyloidosis by gadolinium cardiac magnetic resonance
Enrica Perugini,Claudio Rapezzi,Tommaso Piva,Ornella Leone,Letizia Bacchi-Reggiani,Letizia Riva,Fabrizio Salvi,Luigi Lovato,Angelo Branzi,Rossella Fattori +9 more
TL;DR: Gd enhancement is common but not universally present in patients with cardiac amyloidosis, probably due to expansion of infiltrated interstitium and appears to be associated with impaired segmental and global contractility and a larger atrium.
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Assessment of white matter tract damage in patients with amyotrophic lateral sclerosis: a diffusion tensor MR imaging tractography study.
Federica Agosta,Elisabetta Pagani,Melissa Petrolini,Domenico Caputo,M. Perini,Alessandro Prelle,Fabrizio Salvi,Massimo Filippi +7 more
TL;DR: The association of CST damage with the rate of disease progression suggests that DTI has the potential to provide in vivo markers of ALS evolution and the subtle involvement of the uncinate fasciculus may precede the appearance of behavioral symptoms in patients with ALS.
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Double-blind study of vigabatrin in the treatment of drug-resistant epilepsy.
TL;DR: The results demonstrate the efficacy and good tolerability of vigabatrin therapy in patients with severe complex partial epilepsy and show a significant reduction in seizure frequency during treatment.
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Cardiac amyloidosis: the great pretender
Claudio Rapezzi,Massimiliano Lorenzini,Simone Longhi,Agnese Milandri,Christian Gagliardi,Ilaria Bartolomei,Fabrizio Salvi,Mathew S. Maurer +7 more
TL;DR: The awareness of this condition is the most important prerequisite for the management of the risk of underdiagnoses and misdiagnosis, but fortunately each of these diagnostic steps can also offer diagnostic “red flags” (i.e. highly suggestive findings that can foster the correct diagnostic suspicion and facilitate early, timely diagnosis).
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Two Italian kindreds with familial amyotrophic lateral sclerosis due to FUS mutation
Adriano Chiò,Gabriella Restagno,Maura Brunetti,Irene Ossola,Andrea Calvo,Gabriele Mora,Mario Sabatelli,Maria Rosaria Monsurrò,Stefania Battistini,Jessica Mandrioli,Fabrizio Salvi,Rossella Spataro,Jennifer C. Schymick,Bryan J. Traynor,Vincenzo La Bella +14 more
TL;DR: FUS/TLS gene, located on chromosome 16p11.2, has been identified as a disease gene in familial amyotrophic lateral sclerosis (FALS) in a cohort of 52 index cases from seven Italian regions with non-SOD1 and non-TARDBP FALS.