F
Fei Sun
Researcher at Shanghai Jiao Tong University
Publications - 64
Citations - 1978
Fei Sun is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Spermatogenesis & Biology. The author has an hindex of 20, co-authored 52 publications receiving 1630 citations. Previous affiliations of Fei Sun include University of Science and Technology of China & Chinese Ministry of Education.
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MicroRNA-224 Is Involved in Transforming Growth Factor-β-Mediated Mouse Granulosa Cell Proliferation and Granulosa Cell Function by Targeting Smad4
TL;DR: This is the first demonstration that miRNAs can control reproductive functions resulting in promoting TGF-beta1-induced GC proliferation and ovarian estrogen release.
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Altered microRNA expression in patients with non-obstructive azoospermia.
TL;DR: This is the first report that the expression of miRNA expression is altered in testicular tissues of patients with NOA, suggesting a role of miRNAs in regulating spermatogenesis in human males.
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Downregulation of microRNA-383 is associated with male infertility and promotes testicular embryonal carcinoma cell proliferation by targeting IRF1.
TL;DR: It is shown that downregulation of miR-383 was associated with hyperactive proliferation of germ cells in patients with mixed patterns of MA and functions as a negative regulator of proliferation by targeting IRF1, in part, through inactivation of the pRb pathway.
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Transactivation of microRNA-383 by steroidogenic factor-1 promotes estradiol release from mouse ovarian granulosa cells by targeting RBMS1
TL;DR: The results suggest that miR-383 functions to promote steroidogenesis by targeting RBMS1, at least in part, through inactivation of c-Myc, and SF-1 acts as a positive regulator of miR's processing and function in GC.
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Transactivation of MicroRNA-320 by MicroRNA-383 Regulates Granulosa Cell Functions by Targeting E2F1 and SF-1 Proteins
TL;DR: These data demonstrated that miR-320 regulates the proliferation and steroid production by targeting E2F1 and SF-1 in the follicular development, and will potentiate the usefulness of miRNA in the treatment of reproduction and some steroid-related disorders.