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Fuller W. Bazer

Researcher at Texas A&M University

Publications -  833
Citations -  50274

Fuller W. Bazer is an academic researcher from Texas A&M University. The author has contributed to research in topics: Conceptus & Endometrium. The author has an hindex of 109, co-authored 803 publications receiving 46194 citations. Previous affiliations of Fuller W. Bazer include Texas A&M University System & Auburn University.

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Interferon Tau in the Ovine Uterus

TL;DR: The history, signaling pathways of IFNT and the uterine expression of several IFNT-stimulated genes during the peri-implantation period are reviewed, believed to be critical to unraveling the mechanism(s) of reciprocal fetal-maternal interactions required for successful implantation and pregnancy.
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Placental adaptation to maternal malnutrition.

TL;DR: A review of placental responses to maternal undernutrition related to changes in placental vascularization and hemodynamics and placental nutrient transport systems across species can be found in this article.
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Metabolic pathways utilized by the porcine conceptus, uterus, and placenta.

TL;DR: The synthesis of ATP, nucleotides, amino acids, and fatty acids through these biosynthetic pathways is essential to support elongation, migration, hormone synthesis, implantation, and early placental development of conceptuses.
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C-C motif chemokine ligand 23 abolishes ER stress- and LPS-induced reduction in proliferation of bovine endometrial epithelial cells.

TL;DR: CCL23 improves endometrial development and uterine receptivity required for implantation and placentation during early pregnancy by inhibiting the expression of pro‐inflammatory cytokines and restoring intracellular Ca2+ levels.
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Effect of supplementation of unprotected or protected arginine to prolific ewes on maternal amino acids profile, lamb survival at birth, and pre- and post-weaning lamb growth.

TL;DR: The hypothesis was that rumen protected arginine, the precursor for nitric oxide and polyamines, would increase placental development and vascularity, utero-placental blood flow and nutrient transport, and reduce oxidative stress to increase LSRAB.