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G. J. A. Offerhaus

Researcher at Johns Hopkins University

Publications -  5
Citations -  1030

G. J. A. Offerhaus is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Cancer & Gastrectomy. The author has an hindex of 5, co-authored 5 publications receiving 1008 citations. Previous affiliations of G. J. A. Offerhaus include Leiden University.

Papers
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Journal ArticleDOI

Increased Risk of Cancer in the Peutz–Jeghers Syndrome

TL;DR: It is suggested that patients with the Peutz-Jeghers syndrome have an increased risk for the development of cancer at gastrointestinal and nongastrointestinal sites.
Journal Article

Meta-analysis of the risk of gastric stump cancer: detection of high risk patient subsets for stomach cancer after remote partial gastrectomy for benign conditions.

TL;DR: Although differences in risk between subsets of postagastrectomy patients seen to exist, recommendations concerning endoscopic surveillance await further studies of cost-benefit analysis.
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Multivariate Analysis of the Risk of Stomach Cancer after Ulcer Surgery in an Amsterdam Cohort of Postgastrectomy Patients

TL;DR: In this paper, the authors analyzed risk factors for stomach cancer mortality in an Amsterdam cohort of 2,633 postgastrectomy patients, followed from their initial surgery between 1931 and 1960 until 1975, with 99.7% complete followup.
Journal Article

Gastric, pancreatic, and colorectal carcinogenesis following remote peptic ulcer surgery. Review of the literature with the emphasis on risk assessment and underlying mechanism.

TL;DR: In the long run, the hypochlorhydric stomach might be a condition suitable for the chemoprevention of cancer and further research of the putative relationship between peptic ulcer surgery and carcinogenesis and their underlying mechanism is highly desirable.
Journal ArticleDOI

Occurrence of non-gastric cancer in the digestive tract after remote partial gastrectomy: analysis of an Amsterdam cohort.

TL;DR: The hypothesis that carcinogens are not only locally activated in the gastric remnant, but are hepatically excreted and initiate cancer in the biliary tree and pancreatic duct is supported.