G
Georg Hagemann
Researcher at Schiller International University
Publications - 65
Citations - 3637
Georg Hagemann is an academic researcher from Schiller International University. The author has contributed to research in topics: Cerebral cortex & Lesion. The author has an hindex of 31, co-authored 65 publications receiving 3356 citations. Previous affiliations of Georg Hagemann include University of Jena & University of Düsseldorf.
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Journal ArticleDOI
Anticoagulant reversal, blood pressure levels, and anticoagulant resumption in patients with anticoagulation-related intracerebral hemorrhage.
Joji B. Kuramatsu,Stefan T. Gerner,Peter D. Schellinger,Jörg Glahn,Matthias Endres,Jan Sobesky,Julia Flechsenhar,Hermann Neugebauer,Hermann Neugebauer,Eric Jüttler,Eric Jüttler,Armin J. Grau,Frederick Palm,Joachim Röther,Peter Michels,Gerhard F. Hamann,Joachim Hüwel,Georg Hagemann,Beatrice Barber,Christoph Terborg,Frank Trostdorf,Hansjörg Bäzner,Aletta Roth,Johannes C. Wöhrle,Moritz Keller,Michael P. Schwarz,Gernot Reimann,Jens Volkmann,Wolfgang Müllges,Peter Kraft,Joseph Classen,Carsten Hobohm,Markus Horn,Angelika Milewski,Heinz Reichmann,Hauke Schneider,Eik Schimmel,Gereon R. Fink,Christian Dohmen,Henning Stetefeld,Otto W. Witte,Albrecht Günther,Tobias Neumann-Haefelin,Andras E. Racs,Martin Nueckel,Frank Erbguth,Stephan P. Kloska,Arnd Dörfler,Martin Köhrmann,Stefan Schwab,Hagen B. Huttner +50 more
TL;DR: Among patients with OAC-associated ICH, reversal of anticoagulation reversal and blood pressure (BP) with hematoma enlargement and resumption of OAC therapy was associated with lower risk of ischemic events, and these findings require replication and assessment in prospective studies.
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Neuronal hyperexcitability and reduction of GABAA-receptor expression in the surround of cerebral photothrombosis.
Klaus Schiene,Claus Bruehl,Karl Zilles,Meishu Qü,Georg Hagemann,Matthias Kraemer,Otto W. Witte +6 more
TL;DR: It is concluded that cortical infarction due to photothrombosis leads to a long-lasting and widespread reduction of GABAA-receptor expression in the surround of the lesion, which is associated with an increased neuronal excitability.
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Functional differentiation of multiple perilesional zones after focal cerebral ischemia.
TL;DR: A broad differentiation of separate topographic perilesional areas according to their functional state and sequelae allows segregation into several signaling cascades, and may help to understand the functional consequences and adaptive processes after focal brain ischemia.
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Increased long-term potentiation in the surround of experimentally induced focal cortical infarction
TL;DR: The results provide further evidence for the lesion‐induced amplification of network plasticity, as it is required for the reshaping of cortical circuits by timely training procedures.
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Electrophysiological Transcortical Diaschisis After Cortical Photothrombosis in Rat Brain
TL;DR: The results suggest that a neocortical infarction leads to hyperexcitability not only in its direct vicinity but also in the contralateral hemisphere and may contribute to increased activation of contral lateral brain areas and to functional reorganization after stroke.