G
Giuseppe N. Colasurdo
Researcher at University of Texas Health Science Center at Houston
Publications - 51
Citations - 1539
Giuseppe N. Colasurdo is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Bronchoalveolar lavage & Lung. The author has an hindex of 21, co-authored 47 publications receiving 1474 citations. Previous affiliations of Giuseppe N. Colasurdo include University of Melbourne & University of Texas at Austin.
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Journal ArticleDOI
Antioxidant treatment ameliorates respiratory syncytial virus-induced disease and lung inflammation.
Shawn M. Castro,Antonieta Guerrero-Plata,Giovanni Suarez-Real,Patrick A. Adegboyega,Giuseppe N. Colasurdo,Amir M. Khan,Roberto P. Garofalo,Antonella Casola +7 more
TL;DR: Modulation of oxidative stress represents a potential novel pharmacologic approach to ameliorate RSV-induced acute lung inflammation and potentially prevent long-term consequences associated with RSV infection, such as bronchial asthma.
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Adenosine-dependent airway inflammation and hyperresponsiveness in partially adenosine deaminase-deficient mice.
Janci L. Chunn,Hays W. J. Young,Suman K. Banerjee,Giuseppe N. Colasurdo,Michael R. Blackburn +4 more
TL;DR: Elevations in adenosine can directly impact lung inflammation and physiology, and this finding may have important implications in the regulation of pulmonary inflammation and airway hyperresponsiveness.
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Isolation of Stachybotrys from the lung of a child with pulmonary hemosiderosis.
TL;DR: This case is reported the first case in which Stachybotrys was isolated from the bronchoalveolar lavage fluid of a child with pulmonary hemorrhage and recovered completely after his immediate removal from the environment and subsequent cleaning of his home.
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Decreased bacterial clearance from the lungs of mice following primary respiratory syncytial virus infection.
TL;DR: It is demonstrated that RSV infection decreases bacterial clearance, potentially predisposing to secondary bacterial pneumonia despite increased lung cellular inflammation, and suggested that functional changes occur in the recruited neutrophils that may contribute to the decreased bacterial clearance.
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Time course of hemosiderin production by alveolar macrophages in a murine model
TL;DR: The findings demonstrate the validity of the Balb/c mice model for the study of HLM production after blood aspiration and evaluate the effect of a single blood aspiration on the recruitment of inflammatory cells within the BAL.