H
Hamdy Awad
Researcher at Ohio State University
Publications - 74
Citations - 1543
Hamdy Awad is an academic researcher from Ohio State University. The author has contributed to research in topics: Aortic aneurysm & Spinal cord injury. The author has an hindex of 17, co-authored 74 publications receiving 1225 citations. Previous affiliations of Hamdy Awad include The Ohio State University Wexner Medical Center & Cleveland Clinic.
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Journal ArticleDOI
The evolving management of acute right-sided heart failure in cardiac transplant recipients.
TL;DR: Only through careful preoperative planning can this life-threatening condition be managed in the postoperative period.
Journal ArticleDOI
The effects of steep trendelenburg positioning on intraocular pressure during robotic radical prostatectomy.
Hamdy Awad,Scott Santilli,Matthew Ohr,Andrew G. Roth,Wendy Yan,Soledad Fernandez,Steven Roth,Vipul R. Patel +7 more
TL;DR: IOP reached peak levels at the end of steep Trendelenburg position (T5), on average 13 mm Hg higher than the preanesthesia induction (T1) value, whereas age, body mass index, blood loss, volume of IV fluid administered, mean airway pressure, and desflurane concentration were not predictive.
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Inflammatory Response and Chemokine Expression in the White Matter Corpus Callosum and Gray Matter Cortex Region During Cuprizone-Induced Demyelination
TL;DR: A model to study early microglia activation and to investigate differences in the neuroinflammatory response of white vs. gray matter is defined and it is clearly able to demonstrate that both regions are characterized by early oligodendrocyte stress/apoptosis with concomitant microglio-astrocytosis.
Journal ArticleDOI
Spinal cord injury after thoracic endovascular aortic aneurysm repair
Hamdy Awad,Mohamed Ehab Ramadan,Mohamed Ehab Ramadan,Hosam F. El Sayed,Daniel A. Tolpin,Esmerina Tili,Charles D. Collard +6 more
TL;DR: This narrative review article is to summarize the current literature on the risk factors for and pathophysiology of spinal cord injury (SCI) following TEVAR, and to discuss various intraoperative monitoring and treatment strategies.
Journal ArticleDOI
Endothelial cell damage is the central part of COVID-19 and a mouse model induced by injection of the S1 subunit of the spike protein.
Gerard J. Nuovo,Cynthia M. Magro,Toni Shaffer,Hamdy Awad,David Suster,Sheridan Mikhail,Bing He,Jean Jacques Michaille,Benjamin Liechty,Esmerina Tili +9 more
TL;DR: It is concluded that ACE2+ endothelial damage is a central part of SARS-CoV2 pathology and may be induced by the spike protein alone.