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Harry A. Quigley

Researcher at Johns Hopkins University

Publications -  506
Citations -  72465

Harry A. Quigley is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Glaucoma & Intraocular pressure. The author has an hindex of 115, co-authored 492 publications receiving 65186 citations. Previous affiliations of Harry A. Quigley include University of Miami & Johns Hopkins University School of Medicine.

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Clinicopathologic features of Chandler's syndrome

TL;DR: The clinicopathologic features of Chandler's syndrome are elucidated based on a study of nine patients, and abnormalities of the endothelially-derived cells lining the posterior corneal surface, the inner surface of the trabecular meshwork, and the anterior iris surface are identified.
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Prolonged Blockade of VEGF Family Members Does Not Cause Identifiable Damage to Retinal Neurons or Vessels

TL;DR: Estimation of the effects of constant blockade of VEGF for up to 7 months has no identifiable deleterious effects on the retina or choroid and support the use of V EGF antagonists in the treatment of retinal diseases.
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Calibration of the TonoLab Tonometer in Mice with Spontaneous or Experimental Glaucoma

TL;DR: The TonoLab accurately reflects IOP in both normal mice and in eyes of mice with experimental or spontaneous glaucoma, with no detectable effect of age.
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The relationship between optic disc area and open-angle glaucoma: the Baltimore Eye Survey.

TL;DR: Disc area was somewhat larger among patients with glaucoma than control subjects, and in a regression model adjusting for age, gender, and race, the significance of this difference had a probability of 0.06.
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Reappraisal of the mechanisms of glaucomatous optic nerve damage.

TL;DR: It is deceptive to define glaucoma solely by field test results or to use a different diagnostic term (ocular hyper­ tension) for persons with normal fields but ele­ vated lOP, as this brief report will sum­ marise the recent additions to information about how elevated lOP causes optic nerve damage.