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Harry A. Quigley

Researcher at Johns Hopkins University

Publications -  506
Citations -  72465

Harry A. Quigley is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Glaucoma & Intraocular pressure. The author has an hindex of 115, co-authored 492 publications receiving 65186 citations. Previous affiliations of Harry A. Quigley include University of Miami & Johns Hopkins University School of Medicine.

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Glaucoma-related Changes in the Mechanical Properties and Collagen Micro-architecture of the Human Sclera.

TL;DR: The observed biomechanical and microstructural changes could be the result of tissue remodeling occuring in glaucoma and are likely to alter the mechanical environment of the optic nerve head and contribute to axonal damage.
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Characteristics of progenitor cells derived from adult ciliary body in mouse, rat, and human eyes.

TL;DR: Progenitor cells from adult mammalian ciliary body have significant, but limited, proliferation potential and express markers characteristic of other progenitor cells and seen during early retinal development.
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Apraclonidine. A one-week dose-response study.

TL;DR: A double-masked, cross-over, dose-response study of apraclonidine hydrochloride in 20 patients with elevated intraocular pressure, finding that the symptoms may be dose-dependent and the pupillary effect was minimal.
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Scanning and transmission electron microscopic studies of two cases of pigment dispersion syndrome

TL;DR: The increased pigment within the trabecular meshwork and Schlemm's canal may indicate that the glaucoma sometimes associated with pigment dispersion is caused by pigmentary obstruction, although congenital imperfections of the outflow channels may also be a factor.
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Change in choroidal thickness and axial length with change in intraocular pressure after trabeculectomy.

TL;DR: The dynamic relationship between change in IOP and the state of sclera and choroid was confirmed by sequential measurements in postoperative trabeculectomy patients, providing estimates of the magnitude of choroidal swelling and scleral volume decrease with IOP lowering.