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Harry A. Quigley

Researcher at Johns Hopkins University

Publications -  506
Citations -  72465

Harry A. Quigley is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Glaucoma & Intraocular pressure. The author has an hindex of 115, co-authored 492 publications receiving 65186 citations. Previous affiliations of Harry A. Quigley include University of Miami & Johns Hopkins University School of Medicine.

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The Size and Shape of the Optic Disc in Normal Human Eyes

TL;DR: The size, shape, and configuration of connective tissue of the optic disc in normal eye-bank eyes from 60 adults showed that the optic discs of blacks were larger and more oval than those of whites, though the axial lengths of the eyes in the two groups were not significantly different.
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Quantitative detection of glaucomatous damage at the posterior pole by retinal thickness mapping: A pilot study

TL;DR: Mapping of the retinal thickness may provide a sensitive method for the detection and monitoring of early glaucomatous tissue loss in the posterior pole, which is unique due to the combination of the direct measurement of neuroretinal loss inThe central field of vision; the mapping capability; and the rapid image acquisition.
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The effect of age on normal human optic nerve fiber number and diameter

TL;DR: The authors found a large variability of axonal number among their patients, and suspect that the explanation is a selective loss of large nerve fibers.
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Prevalence of glaucoma in a rural East African population.

TL;DR: The high prevalence of OAG in this group was similar to that of African-derived persons in the United States but less than in African-Caribbean populations, and ACG was more prevalent in east Africans than suggested by anecdotal reports.
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Biomechanics of the Human Posterior Sclera: Age- and Glaucoma-Related Changes Measured Using Inflation Testing

TL;DR: Differences in the biomechanical response of normal and glaucoma sclera may represent baseline properties that contribute to axon damage, or may be characteristics that result fromglaucomatous disease.