H
Harry A. Quigley
Researcher at Johns Hopkins University
Publications - 506
Citations - 72465
Harry A. Quigley is an academic researcher from Johns Hopkins University. The author has contributed to research in topics: Glaucoma & Intraocular pressure. The author has an hindex of 115, co-authored 492 publications receiving 65186 citations. Previous affiliations of Harry A. Quigley include University of Miami & Johns Hopkins University School of Medicine.
Papers
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Journal ArticleDOI
Manometric calibration and comparison of TonoLab and TonoPen tonometers in rats with experimental glaucoma and in normal mice.
TL;DR: In mouse and rat eyes, including rats with chronic IOP elevation, the TonoLab accurately reflected manometrically set intraocular pressure in an efficient manner.
Journal Article
A self-assessment instrument designed for measuring independent mobility in RP patients: generalizability to glaucoma patients.
TL;DR: The instrument developed for patients with RP, to determine difficulty across a range of mobility situations, is a valid measure of perceived ability for independent mobility in patients with glaucoma.
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Medical Management of a High Bleb Phase after Trabeculectomies
TL;DR: In eyes with this syndrome, the authors used IOP-loweringeyedrops and/or pills, along with continued corticosteroid eyedrops, and in 14 all medications were ultimately discontinued.
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Aqueous flow through the iris-lens channel: estimates of differential pressure between the anterior and posterior chambers.
David M. Silver,Harry A. Quigley +1 more
TL;DR: The flow of aqueous through the iris-lens channel is driven by the pressure differential between the posterior and anterior chambers, and viscous forces within the aQueous govern the magnitudes of the flow resistance and thepressure differential.
Journal ArticleDOI
Optic disc morphology in open-angle glaucoma compared with anterior ischemic optic neuropathies.
Helen V. Danesh-Meyer,Michael V. Boland,Peter J. Savino,Neil R. Miller,Prem S. Subramanian,Christopher A. Girkin,Harry A. Quigley +6 more
TL;DR: NAION and AAION cause loss of R GCs, but have significantly different disc topography compared with OAG at a given level of RGC loss, as measured by nerve fiber layer (NFL) thickness and average visual field loss.